|
| Status |
Public on Mar 02, 2015 |
| Title |
Phenotype specific analyses of p53 reveal distinct regulatory mechanism for chronically activated p53 |
| Organism |
Homo sapiens |
| Experiment type |
Genome binding/occupancy profiling by high throughput sequencing
|
| Summary |
p53 ChIP seq and Histone mark ChIP Seq data in IMR90 (normal human diploid fibroblasts)
|
| |
|
| Overall design |
Phenotypes were created by overexpression of RasG12V, E1A/RasG12V in IMR90 cells, growing IMR90 and 24 hour Etoposide treated IMR90 cells were subjected to p53 and histone mark ChIP Seq
|
| |
|
| Contributor(s) |
Kirschner K, Samarajiwa S, Menon S |
| Citation(s) |
25790137 |
| |
| Submission date |
Dec 19, 2013 |
| Last update date |
May 15, 2019 |
| Contact name |
Chandra Chilamakuri |
| E-mail(s) |
datasubmissions@cruk.cam.ac.uk
|
| Organization name |
Cancer Research UK Cambridge Institute
|
| Street address |
Robinson Way
|
| City |
Cambridge |
| ZIP/Postal code |
CB2 0RE |
| Country |
United Kingdom |
| |
|
| Platforms (2) |
| GPL10999 |
Illumina Genome Analyzer IIx (Homo sapiens) |
| GPL11154 |
Illumina HiSeq 2000 (Homo sapiens) |
|
| Samples (27)
|
|
| Relations |
| BioProject |
PRJNA232127 |
| SRA |
SRP034623 |
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