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Status |
Public on Jul 17, 2024 |
Title |
ARL11 Stable Expression in NHU-hTERT cells |
Organism |
Homo sapiens |
Experiment type |
Expression profiling by array
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Summary |
Analyses of the whole genome mRNA gene expression profiling performed on the normal urothelium cells with stably expressed ARL11 following serum starvation. The results revealed that TP53, RB1 and CDKN2A pathways were activated and the E2F1 and MYC pathways wer inhibitedin cells expressing ARL11. Cells expressing ARL11 also inhibited RAS pathway activation.
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Overall design |
Total RNA was extracted from NHU-hTERT cells transducted with ARL11 and empty vector
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Contributor(s) |
Lee S, Czerniak B, Majewski T |
Citation missing |
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Submission date |
Jul 18, 2013 |
Last update date |
Jul 17, 2024 |
Contact name |
Bogdan Czerniak |
E-mail(s) |
bczernia@mdanderson.org
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Organization name |
UT MD Anderson Cancer Center
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Department |
Pathology
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Street address |
1515 Holcombe
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City |
Houston |
State/province |
TX |
ZIP/Postal code |
77030 |
Country |
USA |
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Platforms (1) |
GPL10558 |
Illumina HumanHT-12 V4.0 expression beadchip |
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Samples (6)
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GSM1192482 |
NHU-hTERT cells with Stable expression of ARL11 after 48 hours A |
GSM1192483 |
NHU-hTERT cells with Stable expression of ARL11 after 48 hours B |
GSM1192484 |
NHU-hTERT cells with Stable expression of ARL11 after 48 hours C |
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Relations |
BioProject |
PRJNA213159 |