NCBI Logo
GEO Logo
   NCBI > GEO > Accession DisplayHelp Not logged in | LoginHelp
GEO help: Mouse over screen elements for information.
          Go
Series GSE253715 Query DataSets for GSE253715
Status Public on Aug 09, 2024
Title RAS-mutant AML LSCs originate from GMPs and drive clinical resistance to BH3 mimetics
Organism Homo sapiens
Experiment type Expression profiling by high throughput sequencing
Genome binding/occupancy profiling by high throughput sequencing
Summary This SuperSeries is composed of the SubSeries listed below.
 
Overall design Refer to individual Series
 
Citation(s) 39478230
Submission date Jan 19, 2024
Last update date Nov 08, 2024
Contact name BiNGS Core
E-mail(s) bings.analytics@gmail.com
Organization name Icahn School of Medicine at Mount Sinai
Street address 1470 Madison Ave
City New York
ZIP/Postal code 10029
Country USA
 
Platforms (2)
GPL18573 Illumina NextSeq 500 (Homo sapiens)
GPL24676 Illumina NovaSeq 6000 (Homo sapiens)
Samples (41)
GSM7496813 AML-4-10-PDX
GSM7496814 AML-4-24-PDX
GSM8020595 R+Ctrl1
This SuperSeries is composed of the following SubSeries:
GSE235206 RAS-mutant AML stem cells acquire resistance to BH3 mimetics through altered BCL-2 family expression
GSE253426 RAS-mutant AML LSCs originate from GMPs and drive clinical resistance to BH3 mimetics [RNA-Seq]
GSE253696 RAS-mutant AML LSCs originate from GMPs and drive clinical resistance to BH3 mimetics [ATAC-Seq]
Relations
BioProject PRJNA1066799

Download family Format
SOFT formatted family file(s) SOFTHelp
MINiML formatted family file(s) MINiMLHelp
Series Matrix File(s) TXTHelp

Supplementary file Size Download File type/resource
GSE253715_RAW.tar 7.7 Gb (http)(custom) TAR (of BW, CSV, MTX, TSV)
SRA Run SelectorHelp
| NLM | NIH | GEO Help | Disclaimer | Accessibility |
NCBI Home NCBI Search NCBI SiteMap