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Status |
Public on Mar 01, 2024 |
Title |
Deficiency of lncRNA MERRICAL abrogates macrophage chemotaxis and alleviates diabetes-associated atherosclerosis |
Organism |
Mus musculus |
Experiment type |
Expression profiling by high throughput sequencing
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Summary |
1) Differential gene expression profiling of bone marrow derived macrophage treated with GapmeR NS (control) or MERRICAL (KD) in both M0 and M1 phase
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Overall design |
M0 and M1 phase bone marrow derived macrophage was treated with GapmeR (NS) and (MERRICAL) for 16 hours, and RNA was collected to compare the transcriptome difference between KD and NS.
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Web link |
https://doi.org/10.1016/j.celrep.2024.113815
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Contributor(s) |
Chen J, Feinberg MW |
Citation(s) |
38428421 |
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Submission date |
Jun 21, 2023 |
Last update date |
Apr 24, 2024 |
Contact name |
Jingshu Chen |
E-mail(s) |
ttpsycho7@gmail.com
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Organization name |
Harvard Medical School
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Department |
Medicine
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Lab |
Feinberg Lab
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Street address |
77 Avenue Louis PasteurBoston
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City |
Boston |
ZIP/Postal code |
02115 |
Country |
USA |
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Platforms (1) |
GPL24247 |
Illumina NovaSeq 6000 (Mus musculus) |
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Samples (11)
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Relations |
BioProject |
PRJNA986050 |