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Status |
Public on Jul 24, 2023 |
Title |
Zfp296 knockout enhances chromatin accessibility and induces a unique state of pluripotency in embryonic stem cells [ATAC-seq] |
Organism |
Mus musculus |
Experiment type |
Genome binding/occupancy profiling by high throughput sequencing
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Summary |
To explore the molecular mechanism underlying transcriptional repression by ZFP296, we performed genome-wide analysis of chromatin accessibility (ATAC-seq).
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Overall design |
ATAC-seq for wild-type, Zfp296-knockout #98, and Zfp296-rescue #22 ESCs.
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Contributor(s) |
Miyazaki J, Miyazaki S, Yamano H, Motooka D |
Citation(s) |
37488353 |
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Submission date |
May 01, 2023 |
Last update date |
Jun 06, 2024 |
Contact name |
Daisuke Motooka |
E-mail(s) |
dry_team@ngs.gen-info.osaka-u.ac.jp
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Organization name |
NGS core facility, Research Institute for Microbial Diseases, Osaka University
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Street address |
3-1, Yamadaoka
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City |
Suita |
State/province |
Osaka |
ZIP/Postal code |
5650871 |
Country |
Japan |
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Platforms (1) |
GPL24247 |
Illumina NovaSeq 6000 (Mus musculus) |
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Samples (3) |
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This SubSeries is part of SuperSeries: |
GSE231412 |
Zfp296 knockout enhances chromatin accessibility and induces a unique state of pluripotency in embryonic stem cells |
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Relations |
BioProject |
PRJNA964780 |