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Status |
Public on May 06, 2022 |
Title |
Manipulating Cholesterol Metabolism by oncolytic virus suppresses metastasis of triple-negative breast cancer [ChIP-Seq] |
Organism |
Mus musculus |
Experiment type |
Genome binding/occupancy profiling by high throughput sequencing
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Summary |
Investigate the role of FOXO3a in the ApoA1 induced KRT14 suppression.
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Overall design |
4T1 with ApoA1-over expression were cultured for 24h. CHIP-seq was performed in 4T1ApoA1 lysis by using Anti-FOXO3a.
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Contributor(s) |
Jie D, Likai K |
Citation missing |
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Submission date |
May 03, 2022 |
Last update date |
May 08, 2022 |
Contact name |
Jie Dong |
E-mail(s) |
dongjie@nju.edu.cn
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Organization name |
Nanjing University
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Street address |
Hankou road 22
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City |
Nanjing |
ZIP/Postal code |
210000 |
Country |
China |
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Platforms (1) |
GPL24247 |
Illumina NovaSeq 6000 (Mus musculus) |
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Samples (2) |
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This SubSeries is part of SuperSeries: |
GSE202428 |
Manipulating Cholesterol Metabolism by oncolytic virus suppresses metastasis of triple-negative breast cancer |
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Relations |
BioProject |
PRJNA834756 |