|
| Status |
Public on Jan 01, 2020 |
| Title |
Genome occupancy of NIPBL in mouse P19 teratocarcinoma cells. |
| Organism |
Mus musculus |
| Experiment type |
Genome binding/occupancy profiling by high throughput sequencing
|
| Summary |
Mutations in NIPBL are the major cause of Cornelia de Lange Syndrome (CdLS). NIPBL is the cohesin loading factor and has recently been associated with the BET (Bromodomains and Extra Terminal (ET) domain) proteins BRD2 and BRD4. Related to this, a CdLS-like phenotype has been described associated to BRD4 mutations. We have study the genomic occupancy of NIPBL in mouse P19 teratocarcinoma cells.
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| |
|
| Overall design |
Chi-seq of Nipbl and imput
|
| |
|
| Contributor(s) |
Luna-Peláez N, Guerrero-Martínez JA, Reyes JC, García-Domínguez M |
| Citation(s) |
31320616 |
| |
| Submission date |
Jun 14, 2019 |
| Last update date |
Apr 01, 2020 |
| Contact name |
Jose C. Reyes |
| E-mail(s) |
jose.reyes@cabimer.es
|
| Phone |
34-954467842
|
| Organization name |
CABIMER
|
| Department |
Genome biology
|
| Street address |
Av. Américo Vespucio
|
| City |
Sevilla |
| ZIP/Postal code |
41092 |
| Country |
Spain |
| |
|
| Platforms (1) |
| GPL19057 |
Illumina NextSeq 500 (Mus musculus) |
|
| Samples (2) |
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| Relations |
| BioProject |
PRJNA548964 |
| SRA |
SRP201496 |
