mDRIP-seq: a high-throughput method for R-loop mapping and quantitative assessment

mDRIP-seq is a high-throughput method for quantitative R-loop landscape profiling

Mitochondrial RNA-seq and total RNA-seq of 293T, HeLa, and N2a cells; RIP-seq of SUPV3L1, ceSUPV3L1, ATP5B, and TRAP1; Small RNA-seq of 293T mitochondrial fractions

Divergent composition and transposon-silencing activity of small RNAs in mammalian oocytes

Balanced SET levels favor the correct enhancer repertoire during cell fate acquisition

Direct DNA crosslinking with CAP-C uncovers transcription-dependent chromatin organization at high resolution

KARR-seq reveals principles of high-order ribonucleoprotein structure assembly

High-resolution transcriptional analysis of immunoglobulin variable regions reveals the absence of direct relationships between somatic hypermutation, nascent transcription and epigenetic marks [PRO-cap]

High-resolution transcriptional analysis of immunoglobulin variable regions reveals the absence of direct relationships between somatic hypermutation, nascent transcription and epigenetic marks

Autonomous transposons tune their sequences to ensure somatic suppression [FLASH]

Autonomous transposons tune their sequences to ensure somatic suppression.

Transcriptome-wide profiling and quantification of N6-methyladenosine by enzyme-assisted adenosine deamination

Joint epigenome profiling reveals cell-type-specific gene regulatory programs in human cortical organoids

ssDRIP-seq: a high-throughput method for R-loop mapping and quantitative assessment

The Dynamic Epigenetic Landscape of the Retina During Development, Reprogramming, and Tumorigenesis

Activation of HOTTIP lncRNA perturbs HSC function leading to AML like disease

HOXBLINC is aberrantly expressed in acute myeloid leukemia and functions as a potent oncogenic long non-coding RNA in leukemogenesis

A neutropenia-associated transcription factor mutation differentially impacts target genes in the cell-states traversed during granulocyte specification and commitment

MAFG-driven astrocytes promote CNS inflammation

Combined Cohesin-Runx1 Deficiency Synergistically Perturbs Chromatin Looping and Causes Myelodysplastic Syndromes [Hi-C]