3D chromatin topography of LSK cells with hyperactivated Notch1 and/or Tcf7 knock-out [HiC-seq]

A STAG2-PAXIP1/PAGR1 axis suppresses lung tumorigenesis

A WIZ/Cohesin/CTCF complex anchors DNA loops to define gene expression and cell identity [Hi-C]

A biophysical basis for the diffusion-restricted behavior of Xist RNA

A cohesin cancer mutation reveals a role for the hinge domain in genome organization and gene expression [Hi-C]

A distal super enhancer mediates estrogen-dependent mouse uterine–specific gene transcription of Insulin-like growth factor 1 (Igf1)

A multiple super-enhancer region establishes inter-TAD interactions and controls Hoxa function in cranial neural crest

A multiscale 3D chromatin architecture that controls development of the humoral immune system is assembled by IKAROS

A multiscale 3D chromatin architecture that controls development of the humoral immune system is assembled by IKAROS [HiC_LpreB_Inducible_deletion]

A multiscale 3D chromatin architecture that controls development of the humoral immune system is assembled by IKAROS [HiC_LpreB_Steadystate]

A multiscale 3D chromatin architecture that controls development of the humoral immune system is assembled by IKAROS [HiChIP_LpreB steady state]

A multiscale 3D chromatin architecture that controls development of the humoral immune system is assembled by IKAROS [HiChIP_LpreB_Inducible]

A primed immune transcriptional program is activated in oligodendroglia in multiple sclerosis [HiChIP]

Active maintenance of CD8+ T cell naïvety through regulation of global genome architecture [Hi-C]

An Intrinsic Disorder Region Controlling Condensation of a Circadian Clock Component and Rhythmic Transcription in the Liver [ChIA-PET]

An Intrinsically Disordered Region Controlling Condensation of a Circadian Clock Component and Rhythmic Transcription in the Liver

BL-HiC analysis of water avoidance stressed BALB/c mice colon epithelial cells

BRWD1 orchestrates chromatin topology by converting static to dynamic cohesin complexes

BRWD1 orchestrates chromatin topology by converting static to dynamic cohesin complexes (Hi-C)

Balanced SET levels favor the correct enhancer repertoire during cell fate acquisition