Opposing lineage specifiers induce a pro-tumor hybrid-identity state in lung adenocarcinoma [ChIP-seq]

BAHCC1 promotes gene expression in neuronal cells by antagonizing SIN3A-HDAC1

Methionine metabolism regulates T cell fate by KCa3.1 activity

Hyper transcription underlines de novo establishment of 3D chromatin architecture in mouse early embryos

Methionine metabolism regulates T cell fate by KCa3.1 activity (ATAC-Seq)

Methionine metabolism regulates T cell fate by KCa3.1 activity (Cut&Run)

IT-scC&T-seq streamlines scalable, parallel profiling of protein-DNA interactions in single cells

MYC couples SWI/SNF and metabolic reprogramming to sustain minimal residual disease in mTORC1-activated cancer cells [CUT&RUN, ATAC-seq]

MYC couples SWI/SNF and metabolic reprogramming to sustain minimal residual disease in mTORC1-activated cancer cells

EGR2 O-GlcNAcylation Orchestrates the Development of Protumoral Macrophages to Limit CD8+ T Cell Antitumor Responses.

The transcription factor IRF4 regulates the differentiation and function of intestinal ILC3s

Cohesin recruitment to promoters and enhancers by general transcription factors and chromatin regulators

Oridonin as a Novel KDM5C Inhibitor Alleviates Clonal Hematopoiesis Induced Cardiac Aging via H3K4me3 Dependent SASP Suppression [ChIP-seq]

SOX2 regulates foregut squamous epithelial homeostasis and is lost during Barrett’s esophagus development [CUT&Run]

Eradicating neuroblastomas by an RBM39 degrader in combination with anti-GD2 immunotherapy regardless of cancer cell state alterations

Creb5 controls its own expression and directly induces the joint-interzone regulatory program [ATACseq_Prx1]

Creb5 controls its own expression and directly induces the joint-interzone regulatory program [ATACseq_Sox9_GFP]

Creb5 controls its own expression and directly induces the joint-interzone regulatory program [CutandRun_Sox9_GFP]

Creb5 controls its own expression and directly induces the joint-interzone regulatory program

We want to check chromotin conformation changes and chromotin accesiblility. We also want to compare gene profile with and without indusilam resistant tumors in mouse genetic model