Innate immunity and Nfkb pathway control prostate stem cell plasticity, reprogramming and tumor initiation

Pbrm1 loss induces a permissive chromatin state for cholangiocytic differentiation and cholangiocarcinoma formation [ATAC-seq]

ARMC5 selectively degrades SCAP-free SREBF1 and is essential for fatty acid desaturation in adipocytes.

ATF4 drives regulatory T cell functional specification in homeostasis and obesity [ATAC-seq]

ATF4 drives regulatory T cell functional specification in homeostasis and obesity [ChIP-seq]

Targeting the chromatin remodeler BAZ2B mitigates liver aging and fibrosis

RNA Cytidine Acetyltransferase NAT10 Maintains T Cell Pathogenicity in Inflammatory Bowel Disease [ATAC-Seq]

Genome binding/occupancy profiling of ALKBH5 by high throughput sequencing [Cut&Tag]

Tracing functional (epi)genomic imprints and their evolutionary origins in human defense antiviral cellular response (ChIP-Seq)

Tracing functional (epi)genomic imprints and their evolutionary origins in human defense antiviral cellular response (ChIP-seq III)

Early Notch signaling programs effector CD8+ T cell differentiation [ATAC-Seq]

TET2 Deficiency Increases the Competitive Advantage of Hematopoietic Stem Cells through Upregulation of Thrombopoietin Receptor Signaling

G-Quadruplex DNA Suppresses Transcriptional Pause Release During DNA Replication

PRC1 and CTCF-Mediated Transition from Poised to Active Chromatin Loops Drives Bivalent Gene Activation

Nuclei isolation from frozen heart tissues through an easy-to-operating, prompt, and inexpensive method for single-nucleus Multiome-seq

Metabolic Promotion of Adult and Aging Skeletal Muscle Regeneration by the Serine Biosynthesis Pathway [Cut & Run]

Comprehensive mapping of epigenetic events during the T-follicular helper-to-T-regulatory type-1 transdifferentiation pathway reveal a global poised state

The glucocorticoid receptor potentiates aldosterone-induced transcription by the mineralocorticoid receptor

MCRS1 sensitizes pancreatic cancer to T cell immunity [ATAC-seq]

Global epigenetic stability of liver-resident invariant natural killer T cells in response to cues capable of inducing transcriptional re-programming