ChIP-seq of activated murine CD8+ T-cells cultured in high (0.625 mg/mL holotransferrin) or low iron (0.001 mg/mL holotransferrin) conditions

Mtc91 - H3K27me3 ChIPmentation of activated murine CD8+ T-cells cultured in high (0.625 mg/mL holotransferrin) or low iron (0.001 mg/mL holotransferrin) conditions

CTCF-RNA interactions orchestrate cell-specific chromatin loop organization [iCLIP2]

LaminB1 DamID, RNA-seq and Genome-wide analysis of replication timing in RIF1 depleted preimplantation mouse embryos

The Hira complex regulates Gli3R-dependent transcription in Hedgehog signaling and medulloblastoma cell growth and migration

A conserved differentiation program facilitates inhibitory neuron production in the developing mouse and human cerebellum

SUMOylated Etv1 establishes tumor-initiating cells during mammary oncogenesis [ChIP-seq]

SUMOylated Etv1 establishes tumor-initiating cells during mammary oncogenesis

ATAC-seq in ZFP661 overexpression mESCs

Continuous expression of TOX safeguards exhausted CD8 T cell epigenetic fate [coassay_TOXKO]

Continuous expression of TOX safeguards exhausted CD8 T cell epigenetic fate [coassay_TOXKO_LCMV_Arm_rechalle]

Ythdf2 Suppresses the 2C-like State in Mouse Embryonic Stem Cells via Cooperation with Cnot1 [MeRIP-seq]

KMT2C Deficiency Drives Luminal Transdifferentiation into Double-Negative Prostate Cancer and Sensitizes to Fatty Acid Synthase Inhibition [scRNA-Seq]

Tracing functional (epi)genomic imprints and their evolutionary origins in human defense antiviral cellular response (ChIP-Seq)

Tracing functional (epi)genomic imprints and their evolutionary origins in human defense antiviral cellular response

Tracing functional (epi)genomic imprints and their evolutionary origins in human defense antiviral cellular response (ChIP-seq III)

Single-Cell 5-Hydroxymethylcytosine Landscapes of Mammalian Early Embryos at Single-Base Resolution

Brain and cancer associated binding domain mutations provide insight into CTCF’s relationship with chromatin and its ability to act as a chromatin organizer (chip-seq)

Binding domain mutations provide insight into CTCF's relationship with chromatin and its contribution to gene regulation (Omni-ATAC)

Binding domain mutations provide insight into CTCF's relationship with chromatin and its contribution to gene regulation (Hi-C)