A dual histone code specifies the binding of heterochromatin protein Rhino to a subset of piRNA source loci [CUT&RUN chromodomains in S2 cells]

A dual histone code specifies the binding of heterochromatin protein Rhino to a subset of piRNA source loci [CUT&RUN histone modifications in S2 cells]

A dual histone code specifies the binding of heterochromatin protein Rhino to a subset of piRNA source loci

Mitochondria iron transporter ABCB8 contributes to -7/del(7q) leukemia through regulating histone methylation

Dynamic modelling of EWS::FLI1 fluctuations reveals molecular determinants of phenotypic tumor plasticity and prognosis in Ewing sarcoma [CUT&RUN]

Dynamic modelling of EWS::FLI1 fluctuations reveals molecular determinants of phenotypic tumor plasticity and prognosis in Ewing sarcoma

Selective interactions at pre-replication complexes categorize baseline and dormant origins

PAX translocations remodel mitochondrial metabolism through altered leucine usage in rhabdomyosarcoma [Cut&Run]

G-quadruplex profiling in complex tissues using single-cell CUT&Tag

Transcription factor cooperativity at a GATA3 tandem DNA sequence determines oncogenic enhancer-mediated activation.

Dynamic Single Cell Transcriptomics Defines Kidney FGF23/KL Bioactivity and Novel Segment-Specific Inflammatory Targets

ANP32E drives vulnerabiliy to ATR inhibitors in Triple Negative Breast Cancer by accumulation of R-loops [CUT&RUN]

ANP32E drives vulnerabiliy to ATR inhibitors in Triple Negative Breast Cancer by accumulation of R-loops [DRIP-seq]

ANP32E drives vulnerabiliy to ATR inhibitors in Triple Negative Breast Cancer by accumulation of R-loops

NAT10 and N4-Acetylcytidine restrain R-loop levels and related inflammatory responses

NAT10 and N4-Acetylcytidine restrain R-loop levels and related inflammatory responses II

Genome-wide CRISPR screen identifies IRF1 and TFAP4 as transcriptional regulators of Galectin-9 in T cell acute lymphoblastic leukemia [ChIP-seq]

Tumor-associated neutrophil precursors impair homologous DNA repair and promote sensitivity to PARP-inhibition [ChIP-seq]

Tumor-associated neutrophil precursors impair homologous DNA repair and promote sensitivity to PARP-inhibition.

Chromatin accessibility contributes to Candida albicans adaptation to echinocandins