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PTTG3P pituitary tumor-transforming 3, pseudogene [ Homo sapiens (human) ]

Gene ID: 26255, updated on 13-May-2022

Summary

Official Symbol
PTTG3Pprovided by HGNC
Official Full Name
pituitary tumor-transforming 3, pseudogeneprovided by HGNC
Primary source
HGNC:HGNC:13422
See related
Ensembl:ENSG00000213005 AllianceGenome:HGNC:13422
Gene type
pseudo
RefSeq status
VALIDATED
Organism
Homo sapiens
Lineage
Eukaryota; Metazoa; Chordata; Craniata; Vertebrata; Euteleostomi; Mammalia; Eutheria; Euarchontoglires; Primates; Haplorrhini; Catarrhini; Hominidae; Homo
Also known as
PTTG3; rcPTTG1
Summary
Predicted to enable SH3 domain binding activity. Predicted to be involved in homologous chromosome segregation and negative regulation of mitotic sister chromatid separation. Predicted to be located in cytoplasm. Predicted to be active in nucleus. [provided by Alliance of Genome Resources, Apr 2022]
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Genomic context

See PTTG3P in Genome Data Viewer
Location:
8q13.1
Exon count:
1
Annotation release Status Assembly Chr Location
110 current GRCh38.p14 (GCF_000001405.40) 8 NC_000008.11 (66767329..66768034, complement)
110 current T2T-CHM13v2.0 (GCF_009914755.1) 8 NC_060932.1 (67193098..67193803, complement)
105.20220307 previous assembly GRCh37.p13 (GCF_000001405.25) 8 NC_000008.10 (67679564..67680269, complement)

Chromosome 8 - NC_000008.11Genomic Context describing neighboring genes Neighboring gene valosin containing protein interacting protein 1 Neighboring gene C8orf44-SGK3 readthrough Neighboring gene chromosome 8 putative open reading frame 44 Neighboring gene serum/glucocorticoid regulated kinase family member 3 Neighboring gene minichromosome maintenance domain containing 2 Neighboring gene small nucleolar RNA, C/D box 87 Neighboring gene small nucleolar RNA host gene 6 Neighboring gene transcription factor 24

Genomic regions, transcripts, and products

Bibliography

GeneRIFs: Gene References Into Functions

What's a GeneRIF?

General gene information

Markers

Gene Ontology Provided by GOA

Function Evidence Code Pubs
enables SH3 domain binding IEA
Inferred from Electronic Annotation
more info
 
Process Evidence Code Pubs
involved_in chromosome organization IEA
Inferred from Electronic Annotation
more info
 
involved_in homologous chromosome segregation IBA
Inferred from Biological aspect of Ancestor
more info
PubMed 
involved_in negative regulation of mitotic sister chromatid separation IBA
Inferred from Biological aspect of Ancestor
more info
PubMed 
Component Evidence Code Pubs
located_in cytoplasm IEA
Inferred from Electronic Annotation
more info
 
is_active_in nucleus IBA
Inferred from Biological aspect of Ancestor
more info
PubMed 

NCBI Reference Sequences (RefSeq)

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RefSeqs maintained independently of Annotated Genomes

These reference sequences exist independently of genome builds. Explain

These reference sequences are curated independently of the genome annotation cycle, so their versions may not match the RefSeq versions in the current genome build. Identify version mismatches by comparing the version of the RefSeq in this section to the one reported in Genomic regions, transcripts, and products above.

RNA

  1. NR_002734.2 RNA Sequence

    Status: VALIDATED

    Source sequence(s)
    AC011031, AF095289
    Related
    ENST00000521862.1

RefSeqs of Annotated Genomes: Homo sapiens Annotation Release 110 details...Open this link in a new tab

The following sections contain reference sequences that belong to a specific genome build. Explain

Reference GRCh38.p14 Primary Assembly

Genomic

  1. NC_000008.11 Reference GRCh38.p14 Primary Assembly

    Range
    66767329..66768034 complement
    Download
    GenBank, FASTA, Sequence Viewer (Graphics)

Alternate T2T-CHM13v2.0

Genomic

  1. NC_060932.1 Alternate T2T-CHM13v2.0

    Range
    67193098..67193803 complement
    Download
    GenBank, FASTA, Sequence Viewer (Graphics)

Suppressed Reference Sequence(s)

The following Reference Sequences have been suppressed. Explain

  1. NM_021000.1: Suppressed sequence

    Description
    NM_021000.1: This RefSeq record was removed by NCBI staff. Contact info@ncbi.nlm.nih.gov for further information.