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Links from GEO DataSets

Items: 8

1.

Virus-Induced Airway Disease in Mice (C57BL/6J, d21/d49)

(Submitter supplied) Analysis of gene expression in lungs of C57BL/6J mice that develop chronic airway disease phenotypes after a single Sendai virus infection, compared with mice treated with UV-inactivated virus. Keywords: disease state analysis
Organism:
Mus musculus
Type:
Expression profiling by array
Platforms:
GPL339 GPL1261 GPL340
18 Samples
Download data: CEL
Series
Accession:
GSE10964
ID:
200010964

PubMed Full text in PMC Similar studies Analyze with GEO2R

2.

Virus-Induced Airway Disease in Mice (C57BL/6J, d3)

(Submitter supplied) Analysis of gene expression in lungs of C57BL/6J mice that develop chronic airway disease phenotypes after a single Sendai virus infection, compared with mice treated with UV-inactivated virus. Keywords: disease state analysis
Organism:
Mus musculus
Type:
Expression profiling by array
Platform:
GPL17543
12 Samples
Download data: TXT
Series
Accession:
GSE61437
ID:
200061437

PubMed Full text in PMC Similar studies Analyze with GEO2R

3.

Virus-Induced Airway Disease in Mice (C57BL/6J, d49)

(Submitter supplied) Analysis of gene expression in lungs of C57BL/6J mice that develop chronic airway disease phenotypes after a single Sendai virus infection, compared with mice treated with UV-inactivated virus. Keywords: disease state analysis
Organism:
Mus musculus
Type:
Expression profiling by array
Platform:
GPL17543
12 Samples
Download data: TXT
Series
Accession:
GSE49603
ID:
200049603

PubMed Full text in PMC Similar studies Analyze with GEO2R

4.

Airway Epithelial Cell Response to Sendai virus infection

(Submitter supplied) Oligonucleotide microarrays were used to establish a profile for gene expression in wild-type airway epithelial cells after paramyxoviral infection. Analysis was performed on mRNA isolated from SeV-infected primary-culture mouse tracheal epithelial cells that were maintained under physiologic conditions (air-liquid interface). Keywords: Treatment Comparison
Organism:
Mus musculus
Type:
Expression profiling by array
Dataset:
GDS3210
Platform:
GPL81
10 Samples
Download data: CEL
Series
Accession:
GSE10211
ID:
200010211

PubMed Similar studies Analyze with GEO2R

5.
Full record GDS3210

Airway epithelial cells response to Sendai virus infection in vitro

Analysis of primary culture tracheal epithelial cells following infection with Sendai virus (SeV), a common paramyxovirus.Respiratory paramyxoviral infections are a leading cause of serious respiratory disease. Results provide insight into the role of epithelial cells in antiviral defense.
Organism:
Mus musculus
Type:
Expression profiling by array, transformed count, 2 infection sets
Platform:
GPL81
Series:
GSE10211
10 Samples
Download data: CEL
DataSet
Accession:
GDS3210
ID:
3210

PubMed Similar studies GEO Profiles Analyze DataSet

6.

RNA-sequencing of bronchial epithelial cells from an adult cohort including asthmatics, COPD and healthy controls, cultured with Rhinovirus 1A

(Submitter supplied) Rhinovirus infections exacerbate chronic respiratory inflammatory diseases such as asthma and chronic obstructive pulmonary disease (COPD). Airway epithelial cells are the primary site of rhinovirus replication and responsible for initiating the host immune response to infection. Numerous studies have reported that the anti-viral innate immune response in asthma is deficient leading to the conclusion that epithelial innate immunity is a key determinant of disease severity during a rhinovirus induced exacerbation. more...
Organism:
Homo sapiens
Type:
Expression profiling by high throughput sequencing
Platform:
GPL11154
120 Samples
Download data: CSV
Series
Accession:
GSE146532
ID:
200146532

PubMed Full text in PMC Similar studies Analyze with GEO2RSRA Run Selector

7.

Cigarette smoke silences innate lymphoid cell function in the lung, facilitating an IL-33-dependent, exacerbated Th-1 response to infection

(Submitter supplied) Using gene expression profiling to examine how IL33 treatment affect gene expression in lung tissues of mice
Organism:
Mus musculus
Type:
Expression profiling by array
Platform:
GPL1261
32 Samples
Download data: CEL
Series
Accession:
GSE66492
ID:
200066492

PubMed Similar studies Analyze with GEO2R

8.

Vitamin A-deficient mice experience increased viral antigens and enhanced cytokine/chemokine production in nasal tissues following respiratory virus infection despite the presence of FoxP3+ T cells

(Submitter supplied) Vitamin A deficiencies and insufficiencies affect hundreds of millions of people in both developing and developed countries. These individuals experience higher rates of mortality and increased morbidity during enteric and respiratory infections compared to those who are vitamin A sufficient. Previously, our laboratory has demonstrated that vitamin A-deficient (VAD) mice have significantly impaired virus-specific IgA and CD8+ T cell responses in the airways. more...
Organism:
Mus musculus
Type:
Expression profiling by array
Platform:
GPL11180
6 Samples
Download data: CEL
Series
Accession:
GSE65805
ID:
200065805

PubMed Full text in PMC Similar studies Analyze with GEO2R

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