GSM3395157[Accession] - GEO DataSets - NCBI

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Items: 4

Select item 2001201641.

Genome-wide CRISPR-Cas9 interrogation of splicing networks reveals a mechanism for recognition of autism-misregulated neuronal microexons [SPAR-seq]

(Submitter supplied) Alternative splicing has critical roles in diverse cellular, developmental and pathological processes. However, the full repertoires of factors that control individual splicing events are not known. We describe a CRISPR-based screening strategy for the systematic identification of genes that control 3-27 nt microexons with functions in nervous system development and that are commonly disrupted in autism. more...

Organism:: Mus musculus

Type:: Other

Platform:: GPL16417
48 Samples

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Series

Accession:: GSE120164

ID:: 200120164

PubMed Similar studies SRA Run Selector

Select item 2001126012.

Genome-wide CRISPR-Cas9 interrogation of splicing networks reveals a mechanism for recognition of autism-misregulated neuronal microexons

(Submitter supplied) This SuperSeries is composed of the SubSeries listed below.

Organism:: synthetic construct; Mus musculus

Type:: Expression profiling by high throughput sequencing; Other

4 related Platforms
122 Samples

Series

Accession:: GSE112601

ID:: 200112601

PubMed Similar studies

Select item 1000164173.

Illumina MiSeq (Mus musculus)

Organism:: Mus musculus

972 Series
22872 Samples

Platform

Accession:: GPL16417

ID:: 100016417

Select item 3033951574.

SPAR-Seq of multi-exon regions of selected genes in N2A, Srrm4+siSrrm3 knockdown (T024), replicate 1

Organism:: Mus musculus

Source name:: Neuroblastoma cells

Platform:: GPL16417
Series:: GSE112601 GSE120164

Sample

Accession:: GSM3395157

ID:: 303395157

SRA Run Selector

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