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Search results: x Variables, w Analyses, x Documents, and z Datasets in x Studies
Clinical VariableDatasetVariable DescriptionVariable ID
The data table contains mapping of study subject IDs to sample IDs. Samples are the final preps submitted for genotyping, sequencing, and/or expression data. For example, if one patient (subject ID) gave one sample, and that sample was processed differently to generate 2 sequencing runs, there would be two rows, both using the same subject ID, but having 2 unique sample IDs. This table also includes sample use.
Sample source IDphv00180635.v1.p1
The data table contains mapping of study subject IDs to sample IDs. Samples are the final preps submitted for genotyping, sequencing, and/or expression data. For example, if one patient (subject ID) gave one sample, and that sample was processed differently to generate 2 sequencing runs, there would be two rows, both using the same subject ID, but having 2 unique sample IDs. This table also includes sample use.
Sample sourcephv00180636.v1.p1
The data table contains mapping of study subject IDs to sample IDs. Samples are the final preps submitted for genotyping, sequencing, and/or expression data. For example, if one patient (subject ID) gave one sample, and that sample was processed differently to generate 2 sequencing runs, there would be two rows, both using the same subject ID, but having 2 unique sample IDs. This table also includes sample use.
Sample IDphv00174302.v1.p1
The subject phenotype data table contains data collected about patient type (n=4 variables; randomized, breakthrough, relapse, and sustained virologic response (SVR)), development of cirrhosis accessed by progression of Ishak fibrosis score (n=4 variables), development of hepatic decompensation (n=16 variables; Child-Turcotte-Pugh (CTP) Score, variceal hemorrhage, ascites, spontaneous bacterial peritonitis, hepatic encephalopathy, development of hepatocellular carcinoma, and death), hepatitis C virus RNA levels (n=3 variables), telomerase activity (TMA), liver transplant status, and hepatic arterial infusion (HAI) therapy status (n=2 variables).
Portal gastropathy M48phv00174328.v1.p1
The subject phenotype data table contains data collected about patient type (n=4 variables; randomized, breakthrough, relapse, and sustained virologic response (SVR)), development of cirrhosis accessed by progression of Ishak fibrosis score (n=4 variables), development of hepatic decompensation (n=16 variables; Child-Turcotte-Pugh (CTP) Score, variceal hemorrhage, ascites, spontaneous bacterial peritonitis, hepatic encephalopathy, development of hepatocellular carcinoma, and death), hepatitis C virus RNA levels (n=3 variables), telomerase activity (TMA), liver transplant status, and hepatic arterial infusion (HAI) therapy status (n=2 variables).
Worsening of baseline varicesphv00174325.v1.p1
The data table contains subject IDs and consent group information.
Subject IDphv00174299.v1.p1
The subject phenotype data table contains data collected about patient type (n=4 variables; randomized, breakthrough, relapse, and sustained virologic response (SVR)), development of cirrhosis accessed by progression of Ishak fibrosis score (n=4 variables), development of hepatic decompensation (n=16 variables; Child-Turcotte-Pugh (CTP) Score, variceal hemorrhage, ascites, spontaneous bacterial peritonitis, hepatic encephalopathy, development of hepatocellular carcinoma, and death), hepatitis C virus RNA levels (n=3 variables), telomerase activity (TMA), liver transplant status, and hepatic arterial infusion (HAI) therapy status (n=2 variables).
Spontaneous bacteria peritonitisphv00174318.v1.p1
The sample attributes data table contains the variables for sample IDs, body part where sample was extracted, sample analyte type, tumor status, and histological type.
Analyte typephv00174341.v1.p1
The subject phenotype data table contains data collected about patient type (n=4 variables; randomized, breakthrough, relapse, and sustained virologic response (SVR)), development of cirrhosis accessed by progression of Ishak fibrosis score (n=4 variables), development of hepatic decompensation (n=16 variables; Child-Turcotte-Pugh (CTP) Score, variceal hemorrhage, ascites, spontaneous bacterial peritonitis, hepatic encephalopathy, development of hepatocellular carcinoma, and death), hepatitis C virus RNA levels (n=3 variables), telomerase activity (TMA), liver transplant status, and hepatic arterial infusion (HAI) therapy status (n=2 variables).
Death all causephv00174315.v1.p1
The subject phenotype data table contains data collected about patient type (n=4 variables; randomized, breakthrough, relapse, and sustained virologic response (SVR)), development of cirrhosis accessed by progression of Ishak fibrosis score (n=4 variables), development of hepatic decompensation (n=16 variables; Child-Turcotte-Pugh (CTP) Score, variceal hemorrhage, ascites, spontaneous bacterial peritonitis, hepatic encephalopathy, development of hepatocellular carcinoma, and death), hepatitis C virus RNA levels (n=3 variables), telomerase activity (TMA), liver transplant status, and hepatic arterial infusion (HAI) therapy status (n=2 variables).
Patient randomized into HALT-C trialphv00174338.v1.p1
The subject phenotype data table contains data collected about patient type (n=4 variables; randomized, breakthrough, relapse, and sustained virologic response (SVR)), development of cirrhosis accessed by progression of Ishak fibrosis score (n=4 variables), development of hepatic decompensation (n=16 variables; Child-Turcotte-Pugh (CTP) Score, variceal hemorrhage, ascites, spontaneous bacterial peritonitis, hepatic encephalopathy, development of hepatocellular carcinoma, and death), hepatitis C virus RNA levels (n=3 variables), telomerase activity (TMA), liver transplant status, and hepatic arterial infusion (HAI) therapy status (n=2 variables).
Genderphv00174312.v1.p1
The subject phenotype data table contains data collected about patient type (n=4 variables; randomized, breakthrough, relapse, and sustained virologic response (SVR)), development of cirrhosis accessed by progression of Ishak fibrosis score (n=4 variables), development of hepatic decompensation (n=16 variables; Child-Turcotte-Pugh (CTP) Score, variceal hemorrhage, ascites, spontaneous bacterial peritonitis, hepatic encephalopathy, development of hepatocellular carcinoma, and death), hepatitis C virus RNA levels (n=3 variables), telomerase activity (TMA), liver transplant status, and hepatic arterial infusion (HAI) therapy status (n=2 variables).
Ishak Fibrosis score at visit M24phv00174309.v1.p1
The subject phenotype data table contains data collected about patient type (n=4 variables; randomized, breakthrough, relapse, and sustained virologic response (SVR)), development of cirrhosis accessed by progression of Ishak fibrosis score (n=4 variables), development of hepatic decompensation (n=16 variables; Child-Turcotte-Pugh (CTP) Score, variceal hemorrhage, ascites, spontaneous bacterial peritonitis, hepatic encephalopathy, development of hepatocellular carcinoma, and death), hepatitis C virus RNA levels (n=3 variables), telomerase activity (TMA), liver transplant status, and hepatic arterial infusion (HAI) therapy status (n=2 variables).
HAI improved by 2+ S00 to M48phv00174331.v1.p1
The sample attributes data table contains the variables for sample IDs, body part where sample was extracted, sample analyte type, tumor status, and histological type.
De-identified sample IDphv00174339.v1.p1
The subject phenotype data table contains data collected about patient type (n=4 variables; randomized, breakthrough, relapse, and sustained virologic response (SVR)), development of cirrhosis accessed by progression of Ishak fibrosis score (n=4 variables), development of hepatic decompensation (n=16 variables; Child-Turcotte-Pugh (CTP) Score, variceal hemorrhage, ascites, spontaneous bacterial peritonitis, hepatic encephalopathy, development of hepatocellular carcinoma, and death), hepatitis C virus RNA levels (n=3 variables), telomerase activity (TMA), liver transplant status, and hepatic arterial infusion (HAI) therapy status (n=2 variables).
Patient type: breakthroughphv00174336.v1.p1
The subject phenotype data table contains data collected about patient type (n=4 variables; randomized, breakthrough, relapse, and sustained virologic response (SVR)), development of cirrhosis accessed by progression of Ishak fibrosis score (n=4 variables), development of hepatic decompensation (n=16 variables; Child-Turcotte-Pugh (CTP) Score, variceal hemorrhage, ascites, spontaneous bacterial peritonitis, hepatic encephalopathy, development of hepatocellular carcinoma, and death), hepatitis C virus RNA levels (n=3 variables), telomerase activity (TMA), liver transplant status, and hepatic arterial infusion (HAI) therapy status (n=2 variables).
HCC developedphv00174307.v1.p1
The subject phenotype data table contains data collected about patient type (n=4 variables; randomized, breakthrough, relapse, and sustained virologic response (SVR)), development of cirrhosis accessed by progression of Ishak fibrosis score (n=4 variables), development of hepatic decompensation (n=16 variables; Child-Turcotte-Pugh (CTP) Score, variceal hemorrhage, ascites, spontaneous bacterial peritonitis, hepatic encephalopathy, development of hepatocellular carcinoma, and death), hepatitis C virus RNA levels (n=3 variables), telomerase activity (TMA), liver transplant status, and hepatic arterial infusion (HAI) therapy status (n=2 variables).
HAI improved by 2+ S00 to M24phv00174330.v1.p1
The subject phenotype data table contains data collected about patient type (n=4 variables; randomized, breakthrough, relapse, and sustained virologic response (SVR)), development of cirrhosis accessed by progression of Ishak fibrosis score (n=4 variables), development of hepatic decompensation (n=16 variables; Child-Turcotte-Pugh (CTP) Score, variceal hemorrhage, ascites, spontaneous bacterial peritonitis, hepatic encephalopathy, development of hepatocellular carcinoma, and death), hepatitis C virus RNA levels (n=3 variables), telomerase activity (TMA), liver transplant status, and hepatic arterial infusion (HAI) therapy status (n=2 variables).
Ishak Fibrosis score at visit M48phv00174310.v1.p1
The subject phenotype data table contains data collected about patient type (n=4 variables; randomized, breakthrough, relapse, and sustained virologic response (SVR)), development of cirrhosis accessed by progression of Ishak fibrosis score (n=4 variables), development of hepatic decompensation (n=16 variables; Child-Turcotte-Pugh (CTP) Score, variceal hemorrhage, ascites, spontaneous bacterial peritonitis, hepatic encephalopathy, development of hepatocellular carcinoma, and death), hepatitis C virus RNA levels (n=3 variables), telomerase activity (TMA), liver transplant status, and hepatic arterial infusion (HAI) therapy status (n=2 variables).
Liver deaths or transplantphv00174333.v1.p1
The data table contains mapping of study subject IDs to sample IDs. Samples are the final preps submitted for genotyping, sequencing, and/or expression data. For example, if one patient (subject ID) gave one sample, and that sample was processed differently to generate 2 sequencing runs, there would be two rows, both using the same subject ID, but having 2 unique sample IDs. This table also includes sample use.
Subject IDphv00174301.v1.p1
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