"Luteal Phase" - Books

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Select item 44036681.

StatPearls [Internet].

Treasure Island (FL): StatPearls Publishing; 2025 Jan-.

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Select item 34012942.

Endotext [Internet].

Feingold KR, Adler RA, Ahmed SF, et al., editors.

South Dartmouth (MA): MDText.com, Inc.; 2000-.

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Select item 26591823.

Database of Abstracts of Reviews of Effects (DARE): Quality-assessed Reviews [Internet].

York (UK): Centre for Reviews and Dissemination (UK); 1995-.

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Select item 52920604.

Endometrial scratch to increase live birth rates in women undergoing first-time in vitro fertilisation: RCT and systematic review.

Metwally M, Chatters R, Pye C, et al.

Southampton (UK): NIHR Journals Library; 2022 Jan. (Health Technology Assessment, No. 26.10.)

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Select item 32905405.

Clinical Applications of Mifepristone (RU 486) and Other Antiprogestins: Assessing the Science and Recommending a Research Agenda.

Institute of Medicine (US) Committee on Antiprogestins: Assessing the Science; Donaldson MS, Dorflinger L, Brown SS, et al., editors.

Washington (DC): National Academies Press (US); 1993.

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Select item 32609806.

Contraceptive Research and Development: Looking to the Future.

Institute of Medicine (US) Committee on Contraceptive Research and Development; Harrison PF, Rosenfield A, editors.

Washington (DC): National Academies Press (US); 1996.

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Select item 31697307.

Biologic Markers in Reproductive Toxicology.

National Research Council (US) Subcommittee on Reproductive and Neurodevelopmental Toxicology.

Washington (DC): National Academies Press (US); 1989.

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Select item 31993528.

Exploring the Biological Contributions to Human Health: Does Sex Matter?

Institute of Medicine (US) Committee on Understanding the Biology of Sex and Gender Differences; Wizemann TM, Pardue ML, editors.

Washington (DC): National Academies Press (US); 2001.

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Select item 32808469.

Medically Assisted Conception: An Agenda for Research.

Institute of Medicine (US) and National Research Council (US) Committee on the Basic Science Foundations of Medically Assisted Conception.

Washington (DC): National Academies Press (US); 1989.

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Select item 141528010.

Madame Curie Bioscience Database [Internet].

Austin (TX): Landes Bioscience; 2000-2013.

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Select item 166869011.

Holland-Frei Cancer Medicine. 6th edition.

Kufe DW, Pollock RE, Weichselbaum RR, et al., editors.

Hamilton (ON): BC Decker; 2003.

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Select item 426697412.

PROMISE: first-trimester progesterone therapy in women with a history of unexplained recurrent miscarriages – a randomised, double-blind, placebo-controlled, international multicentre trial and economic evaluation.

Coomarasamy A, Williams H, Truchanowicz E, et al.

Southampton (UK): NIHR Journals Library; 2016 May. (Health Technology Assessment, No. 20.41.)

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Select item 566123613.

Comparing Essure® and Tubal Ligation to Prevent Pregnancy [Internet].

Gariepy AM, Tancredi DJ, Lewis C, et al.

Washington (DC): Patient-Centered Outcomes Research Institute (PCORI); 2021 Oct.

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Select item 578092614.

Nursing Health Promotion [Internet].

Open Resources for Nursing (Open RN); Ernstmeyer K, Christman E Dr, editors.

Eau Claire (WI): Chippewa Valley Technical College; 2025.

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Select item 512637415.

The Palgrave Handbook of Critical Menstruation Studies [Internet].

Bobel C, Winkler IT, Fahs B, et al., editors.

Singapore: Palgrave Macmillan; 2020.

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Select item 326686416.

Strengthening Research in Academic OB/GYN Departments.

Institute of Medicine (US) Committee on Research Capabilities of Academic Departments of Obstetrics and Gynecology; Townsend J, editor.

Washington (DC): National Academies Press (US); 1992.

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Select item 145639017.

Endocrinology: An Integrated Approach.

Nussey S, Whitehead S.

Oxford: BIOS Scientific Publishers; 2001.

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Select item 244094618.

How Tobacco Smoke Causes Disease: The Biology and Behavioral Basis for Smoking-Attributable Disease: A Report of the Surgeon General.

Centers for Disease Control and Prevention (US); National Center for Chronic Disease Prevention and Health Promotion (US); Office on Smoking and Health (US).

Atlanta (GA): Centers for Disease Control and Prevention (US); 2010.

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Select item 546965219.

Toxicological Profile for Hexachlorobenzene.

Atlanta (GA): Agency for Toxic Substances and Disease Registry (US); 2015 Aug.

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Select item 576900620.

Evaluation of Dietary Protein and Amino Acid Requirements: A Systematic Review [Internet].

Burstad K, Erickson A, Gholizadeh E, et al.

Rockville (MD): Agency for Healthcare Research and Quality (US); 2024 Nov.

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Supplemental Content

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Figure 2: Sagittal view of uterus showing the luteal phase endometrium Contributed by Jenna Kahn, MD

Sonography Gynecology Infertility Assessment, Protocols, and Interpretation.

StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2025 Jan-.
Figure 2.

Inhibin level changes throughout the menstrual cycle.
Inhibin B dominates the follicular phase of the cycle, while Inhibin A dominates the luteal phase.

The Normal Menstrual Cycle and the Control of Ovulation.

Feingold KR, Ahmed SF, Anawalt B, et al., editors. Endotext [Internet]. South Dartmouth (MA): MDText.com, Inc.; 2000-.
Figure 2Explanations for catamenial epilepsy

Top: The estrogen/progesterone hypothesis for catamenial epilepsy is shown schematically. The changes in serum estrogen and progesterone levels during the ovarian cycle are diagrammed for the rat (A) and woman (B). In the rat, there is a four day cycle called the estrous cycle. In women, the 28-day cycle is divided into a follicular and luteal phase. In catamenial epilepsy, the periovulatory and perimenstrual periods are typically when seizures worsen; the analogous times are marked for the rodent and human cycles (72). One of the most common hypotheses to explain catamenial epilepsy is based on the idea that estrogen is proconvulsant and progesterone has the opposite effect (72, 77). In addition, when progesterone levels fall dramatically at the end of the luteal phase, increased excitability has been demonstrated, and this contributes to perimenstrual seizure exacerbation (72, 76, 77, 78, 145, 146).
Bottom: An alternate hypothesis for catamenial epilepsy based on the role of BDNF in actions of estrogen and progesterone. A) The rise in hippocampal BDNF during the estrous cycle in the female rat is shown schematically, based on immunocytochemistry (97), and illustrates a long-lasting elevation in BDNF after the preovulatory surge in estrogen. B) Based on the rodent data, a schematic is shown that illustrates the predicted rise in BDNF levels that would occur in women, i.e., a long-lasting rise in BDNF following the preovulatory rise in serum estrogen levels. Because estrogen also rises during the luteal phase, BDNF could remain elevated for the entire luteal phase and the perimenstrual period. This prediction has support from measurements of serum BDNF in women, which is high during the luteal phase relative to the follicular phase (111). Note that the times when BDNF is elevated and progesterone is low correspond well to the periovulatory and perimenstrual periods, in both rat and woman, providing an alternative hypothesis for catamenial epilepsy.

Temporal Lobe Epilepsy and the BDNF Receptor, TrkB.

Noebels JL, Avoli M, Rogawski MA, et al., editors. Jasper's Basic Mechanisms of the Epilepsies [Internet]. 4th edition. Bethesda (MD): National Center for Biotechnology Information (US); 2012.
Figure 9.

Episodic secretion of LH (top) and progesterone (bottom) during the luteal phase of a woman.
Abbreviations: LH, luteinizing hormone: P, progesterone E2, estradiol; LH + 8, LH surge plus 8 days. (From Filicori M, Butler JP, Crowley WF Jr. Neuroendocrine regulation of the corpus luteum in the human. J Clin Invest. 73:1638 1984.

The Normal Menstrual Cycle and the Control of Ovulation.

Feingold KR, Ahmed SF, Anawalt B, et al., editors. Endotext [Internet]. South Dartmouth (MA): MDText.com, Inc.; 2000-.