"Flavin-Adenine Dinucleotide" - Books

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Select item 44036681.

StatPearls [Internet].

Treasure Island (FL): StatPearls Publishing; 2025 Jan-.

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Select item 14589842.

GeneReviews® [Internet].

Adam MP, Feldman J, Mirzaa GM, et al., editors.

Seattle (WA): University of Washington, Seattle; 1993-2025.

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Select item 29911043.

Dietary Reference Intakes for Thiamin, Riboflavin, Niacin, Vitamin B6, Folate, Vitamin B12, Pantothenic Acid, Biotin, and Choline.

Institute of Medicine (US) Standing Committee on the Scientific Evaluation of Dietary Reference Intakes and its Panel on Folate, Other B Vitamins, and Choline.

Washington (DC): National Academies Press (US); 1998.

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Select item 14152804.

Madame Curie Bioscience Database [Internet].

Austin (TX): Landes Bioscience; 2000-2013.

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Select item 52288985.

Scanning for New Evidence on Riboflavin to Support a Dietary Reference Intake Review.

National Academies of Sciences, Engineering, and Medicine; Health and Medicine Division; Food and Nutrition Board; Committee on Scanning for New Evidence on Riboflavin to Support a Dietary Reference Intake Review; Vorosmarti A, Yaktine AL, Schneeman BO, editors.

Washington (DC): National Academies Press (US); 2021 Jul 28.

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Select item 32530466.

Nutrient Requirements of Laboratory Animals: Fourth Revised Edition, 1995.

National Research Council (US) Subcommittee on Laboratory Animal Nutrition.

Washington (DC): National Academies Press (US); 1995.

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Select item 47956027.

Baculovirus Molecular Biology [Internet]. 4th edition.

Rohrmann GF.

Bethesda (MD): National Center for Biotechnology Information (US); 2019.

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Select item 43240408.

Some Aromatic Amines, Organic Dyes, and Related Exposures.

IARC Working Group on the Evaluation of Carcinogenic Risks to Humans.

Lyon (FR): International Agency for Research on Cancer; 2010. (IARC Monographs on the Evaluation of Carcinogenic Risks to Humans, No. 99.)

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Select item 18813829.

Basic Neurochemistry: Molecular, Cellular and Medical Aspects. 6th edition.

Siegel GJ, Agranoff BW, Albers RW, et al., editors.

Philadelphia: Lippincott-Raven; 1999.

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Select item 518831210.

Trace Metals and Infectious Diseases [Internet].

Nriagu JO, Skaar EP, editors.

Cambridge (MA): MIT Press; 2015.

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Select item 325078811.

Emerging Technologies for Nutrition Research: Potential for Assessing Military Performance Capability.

Institute of Medicine (US) Committee on Military Nutrition Research; Carlson-Newberry SJ, Costello RB, editors.

Washington (DC): National Academies Press (US); 1997.

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Select item 159952412.

The Cell: A Molecular Approach. 2nd edition.

Cooper GM.

Sunderland (MA): Sinauer Associates; 2000.

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Select item 575879813.

Oral Cancer Prevention.

IARC Working Group on the Evaluation of Cancer-Preventive Interventions.

Lyon (FR): International Agency for Research on Cancer; 2023. (IARC Handbooks of Cancer Prevention, No. 19.)

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Select item 444646214.

A Time for Metabolism and Hormones [Internet].

Sassone-Corsi P, Christen Y, editors.

Cham (CH): Springer; 2016.

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Select item 466662215.

Mechanisms of Vascular Disease: A Reference Book for Vascular Specialists [Internet].

Fitridge R, Thompson M, editors.

Adelaide (AU): University of Adelaide Press; 2011.

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Select item 544113216.

Toxicological Profile for Disulfoton.

Atlanta (GA): Agency for Toxic Substances and Disease Registry (US); 2022 Aug.

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Select item 557058417.

Nutrient Requirements of Dairy Cattle: Eighth Revised Edition.

National Academies of Sciences, Engineering, and Medicine; Division on Earth and Life Studies; Board on Agriculture and Natural Resources; Committee on Nutrient Requirements of Dairy Cattle.

Washington (DC): National Academies Press (US); 2021 Aug 30.

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Select item 407287218.

Trichloroethylene, Tetrachloroethylene, and Some Other Chlorinated Agents.

IARC Working Group on the Evaluation of Carcinogenic Risks to Humans.

Lyon (FR): International Agency for Research on Cancer; 2014. (IARC Monographs on the Evaluation of Carcinogenic Risks to Humans, No. 106.)

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Select item 532396219.

Diagnosis and Treatment of Painful Diabetic Peripheral Neuropathy.

Pop-Busui R, Ang L, Boulton AJM, et al.

Arlington (VA): American Diabetes Association; 2022 Feb.

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Select item 538535020.

Introduction to Epigenetics [Internet].

Paro R, Grossniklaus U, Santoro R, et al.

Cham (CH): Springer; 2021.

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Figure 7.16Synthesis of nitric oxide (NO) and related products

Note: FAD = flavin adenine dinucleotide; FMN = flavin mononucleotide; H₄B = tetrahydrobiopterin; H₂O = water; NADP⁺ = glutamate dehydrogenase; NADPH = reduced nicotinamide adenine dinucleotide phosphate; O₂ = oxygen; S-GSNO = S-nitrocysteine.

Pulmonary Diseases.

Centers for Disease Control and Prevention (US); National Center for Chronic Disease Prevention and Health Promotion (US); Office on Smoking and Health (US). How Tobacco Smoke Causes Disease: The Biology and Behavioral Basis for Smoking-Attributable Disease: A Report of the Surgeon General. Atlanta (GA): Centers for Disease Control and Prevention (US); 2010.
Figure 3

Oxygenase and reductase domains of eNOS: binding site or distinct region for calmodulin (CaM), electron transfer control element (ETE), flavin mononucleotide (FMN), flavin adenine dinucleotide (FAD), and NADPH in the reductase domain, and heme, tertahydrobiopterin (BH₄), zinc (Zn), and L-arginine (L-Arg) in the oxygenase domain.

Calreticulin Regulation of Lung Endothelial NOS Activity.

Madame Curie Bioscience Database [Internet]. Austin (TX): Landes Bioscience; 2000-2013.
Fig. 9.9

Flavin adenine dinucleotide (FAD) is a cofactor of lysine demethylase 1 (LSD1). The structure of FAD and its involvement in the histone demethylation mediated by LSD1 are shown. The reaction mediated by LSD1 requires protonation of the nitrogen to be demethylated, which limits LSD1 demethylating abilities to mono- and dimethylated lysines. LSD1-mediated demethylation generates formaldehyde and FADH2. Recycling of FAD occurs via oxidation of FADH2, which produces peroxide

Epigenetics and Metabolism.

Paro R, Grossniklaus U, Santoro R, et al. Introduction to Epigenetics [Internet]. Cham (CH): Springer; 2021.
Figure 34-7

Sources of free radical formation which may contribute to injury during ischemia-reperfusion. Nitric oxide synthase, the mitochondrial electron-transport chain and metabolism of arachidonic acid are among the likely contributors, LOO^•, lipid peroxyl radical; L^•, lipid alkoxyl radical; HETES, hydroxyeicosatetraenoic acids; H₄T, tetrahydrobiopterin; O^•−₂, superoxide radical; CaM, calcium/calmodulin; NO^•, nitric oxide; FAD, flavin adenine dinucleotide; FMN, flavin mononucleotide.

Free Radicals in Hypoxia-Ischemia.

Siegel GJ, Agranoff BW, Albers RW, et al., editors. Basic Neurochemistry: Molecular, Cellular and Medical Aspects. 6th edition. Philadelphia: Lippincott-Raven; 1999.