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StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2024 Jan-.

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Osteopathic Manipulative Treatment: Counterstrain/FPR Procedure - Thoracic Vertebrae

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Last Update: July 19, 2022.

Continuing Education Activity

This procedure outlines thoracic strain-counterstrain and functional positional release. These two indirect techniques are used for somatic dysfunctions of the thoracic spine. The primary indications for using these techniques are back/neck pain and patient intolerance to direct osteopathic techniques or techniques that require the patient to activate musculature. This activity highlights the role of the osteopathic clinician in evaluating and managing patients with musculoskeletal issues and somatic dysfunctions of the thoracic spine.

Objectives:

  • Determine the correct procedure for treating somatic dysfunctions of the thoracic spine with strain-counterstrain and functional position release.
  • Assess the indications/contraindications for treating somatic dysfunctions of the thoracic spine with strain-counterstrain and functional position release.
  • Identify potential complications of strain-counterstrain and functional positional release on the thoracic spine.
  • Communicate the importance of improving care coordination to enhance the delivery of care for patients affected by thoracic segment somatic dysfunctions.
Access free multiple choice questions on this topic.

Introduction

Upper back and neck pain caused by somatic dysfunctions of the thoracic spine are extremely common. Common causes include postural changes and injuries.[1][2] A viscerosomatic response can also cause somatic dysfunctions. For example, patients who present with chest pain may have an underlying cardiac issue and, therefore, might have a corresponding somatic dysfunction at the level of T1-T5 of the spine.

There are a variety of osteopathic manipulative treatments (OMT) for somatic dysfunctions. Indirect osteopathic techniques can frequently treat dysfunctions. Strain-counterstrain (SCS) and Functional Positional Release (FPR) are commonly used indirect techniques. These methods require the clinician to passively take the patient into a position of ease or away from the restrictive barrier. SCS involves placing the patient where the target muscle is shortened, which allows the vertebrae to return to its proper position. FPR uses components of SCS and a functional method with the addition of an activating force (compression or torsion).[3] We discuss these 2 osteopathic techniques to treat somatic dysfunctions of the thoracic spine.

Anatomy and Physiology

There are 12 thoracic vertebrae in the spine, designated as T1-T12.[4] The thoracic vertebrae form a kyphotic curve.[5] It is essential to note the thoracic vertebrae provide attachment to the ribcage.[6] Injuries, specifically of or originating from the ribs, are a common cause of somatic dysfunctions of the thoracic vertebrae due to the articulation between the ribs and thoracic spine.[2] Because of costal facets, the thoracic vertebrae are unique compared to the cervical and lumbar vertebrae.[4]

Each thoracic vertebra has 6 facets to allow for articulation with the ribs.[4] T1, T11, and T12 are distinct from the other thoracic vertebrae. T1 is the only vertebrae where the entire costal facets provide articulation with rib 1, which has a very flat spinous process.[4] In contrast to the other thoracic vertebrae, T11 and T12 do not have facets on the transverse processes.[4]

Lastly, T12 articulates with the lumbar spine at the level of L1. The thoracic vertebrae have several muscular attachments, including erector spinae, interspinales, intertransversarii, latissimus dorsi, multifidus, rhomboid major and minor, rotatores, semispinalis, serratus posterior superior and inferior, splenius capitis and cervicis, and trapezius.[4] The arterial supply of the thoracic spine is primarily from the posterior intercostal arteries, branches from the subclavian artery, and the thoracic aorta.[4] The venous supply is primarily from venous plexuses surrounding the vertebral canal.[4] The nervous supply of the thoracic vertebrae is supplied by the meningeal branches originating from spinal nerves.[4]

SCS and FPR use the body’s intrinsic ability to relax. SCS involves neuromuscular activity between a muscle agonist and antagonist called the Proprioceptive Theory.[7] This theory is based on regulating muscle spindle activity. When lengthening the muscle spindle, there is increased muscle spindle activity and a reflexive muscle contraction, which decreases spindle discharge and reflexive contraction when the muscle shortens.[7] SCS passively shortens the dysfunctional agonist muscle for an extended period, returning the muscle spindle activity to normal.[8] Further studies on similar theories, such as proprioceptive neuromuscular facilitation, demonstrated this mechanism increases the range of motion by improving muscle elasticity.[9]

FPR is similar to SCS. The physiology behind FPR involves altering increased gamma motor neuron activity that affects the muscle spindle.[10] A reduction in the gamma motor neuron activity forces the lengthening of the extrafusal fibers to their relaxed state.[10]

Indications

Indications to perform SCS or FPR on a somatic dysfunction of the thoracic spine include an identified tender point associated with:

  • Back pain
  • Chest pain
  • Neck pain
  • Headache
  • Joint hypo-mobility
  • Fascial restrictions
  • Muscle dysfunction or spasms in the localized area or the thoracic spine.[7]

Indications are also based on the patient’s needs. If the patient is not able to activate their own muscles and needs more assistance from the clinician, both of these passive, indirect techniques are appropriate.

Contraindications

SCS and FPR are contraindicated in any patient who cannot give feedback to the clinician.[7] Contraindications of SCS and FPR include:

  • Acute fracture
  • Shoulder dislocation (specific to thoracic extension somatic dysfunctions)

Equipment

Equipment needed for this procedure includes an exam table, OMT table, or massage table for the patient to sit or lay, and a stool for the clinician to sit.

Personnel

The osteopathic clinician is the only person required to perform these techniques correctly.

Preparation

The clinician should discuss the risks, benefits, and alternative treatment options and obtain informed consent from the patient. Evaluation of the thoracic spine is critical before treatment. Visual assessment, pain scale, muscle strength and range of motion, and identification of tender points (TP) and somatic dysfunction should all occur before initiating treatment.

Technique or Treatment

SCS and FPR are indirect techniques. Indirect techniques require the clinician to place the patient at ease, away from the restrictive barrier [11].

During SCS, the clinician first must locate a tender point (TP), sometimes called Jones tender points. For the thoracic spine, there are 12 anterior (AT1-12), 12 posterior (PT1-12), and 4 lateral (LAT5-8) tender points. Each TP corresponds to a thoracic vertebra; AT1 correlates with thoracic vertebrae T1.

For the anterior TPs 1-8, the patient is supine, and the clinician is seated at the head of the table. For AT 9-12, the patient will be supine, and the clinician will be standing on the side of the patient where the TP is. AT1 is located at the sternal notch. AT2-8 are all located below the sternal notch in a row down the sternum, ending at the xiphoid process. The clinician will modify the patient's position by flexing the patient's head towards the sternum. AT9 is located superior/laterally from the umbilicus, AT10-11 are located inferior/laterally from the umbilicus, and AT12 is located on the superior portion of the iliac crest.

The clinician modifies the patient's position by flexing the patient's hips and knees and resting them on the clinician's leg, which should be positioned on the table. The clinician should then cross the patient's leg farther from the clinician over the patient's other leg to induce side bending and rotating away from the TP. The lateral TP's are located at the costal cartilages of the rib of the corresponding vertebrae. The patient is seated, and the clinician is behind the patient with their leg on the table. The clinician modifies the patient's position by rotating them towards the clinician's leg and away from the TP. The posterior TP's are located at the ends of the transverse processes of the corresponding vertebrae. The patient is prone, and the clinician stands on the TP's opposite side. For the upper TP's, modify by pulling the patient's shoulder back on the same side of the TP. For the lower TP's, modify by pulling the patient's pelvis back on the same side of the TP.

After locating a TP, the clinician asks the patient how they rate their pain at the tender point on a scale of 1 to 10. The clinician modifies the patient's position based on the TP location until the patient reports their pain has decreased to a 3 out of 10 or less on the pain scale. Once the pain is reportedly a 3 out of 10 or less, the clinician holds the modified patient position for 90 seconds. After 90 seconds, the clinician passively returns the patient to neutral.

Before treating the thoracic spine with FPR, a diagnosis of the spine with Fryette's principles is necessary. Fryette's principle says that a group curve is neutral, rotated, and side bent in different directions (eg, T1-T3NRLSR). It also states that if a single vertebra is dysfunctional, it is typically non-neutral (flexed or extended), rotated, and side bent to the same side (eg, T1FRLSL). Since FPR is indirect, place the patient away from the restrictive barrier or into the diagnosis.

The patient is seated, and the clinician is standing behind the patient. Have them sit as straight as possible to straighten the patient's thoracic kyphosis. Ask the patient to cross their arms. The clinician should place their forearm on the opposite shoulder to which the TP is located. The clinician's other hand should be monitoring the thoracic vertebrae of concern. Once the patient and clinician are in the correct position, induce the patient into their diagnosis and add a compressive force with the forearm resting on the patient's shoulder for 3 to 5 seconds.

Complications

SCS and FPR are safe, noninvasive techniques with no serious complications when performed appropriately. After treatment, patients may experience some minor muscle soreness, headache, and lightheadedness. Clinicians should caution their patients regarding soreness lasting 1 to 5 days after treatment.

Clinical Significance

Neck and upper back pain from somatic dysfunctions of the thoracic spine are very common. However, there are limited non-pharmacologic treatment options. SCS and FPR are options that can provide immediate relief. Also, patients who see osteopathic clinicians will receive a holistic and whole-body approach to care. Clinicians treat the thoracic spine and any associated dysfunctions throughout the kinetic chain, including cervical, lumbar, pelvic, and sacral dysfunctions, leading to a better, longer-lasting treatment.

Enhancing Healthcare Team Outcomes

Osteopathic manipulative treatment for somatic dysfunctions of the thoracic spine requires an interprofessional team. Without proper management, acute somatic dysfunctions of the thoracic spine can spiral into severe chronic issues. 

While the primary facilitator of treatment is the osteopathic clinician, a multidisciplinary and holistic approach is now preferred requiring several specialists. In an osteopathic clinic, a resident or fellow clinician practicing neuromuscular medicine gathers information about the patient’s medical, social, and family history and provides pertinent information to the osteopathic clinician. They also assist the osteopathic clinician during the treatment while communicating effectively with the patient.

A pharmacist helps ensure the patient is on the appropriate pain medication and dosage. The musculoskeletal physiotherapist is essential in treating thoracic dysfunctions of the spine. They provide the patient with exercises to strengthen the musculature around the spinal dysfunctions, which the osteopathic clinician addressed.

When the surrounding musculature and spinal segments are addressed, the patient has longer-lasting relief and a better outcome. Lastly, a psychiatrist is a vital component of treating spinal pain according to the biopsychosocial model of care.[12] This model highlights a multidisciplinary approach to persistent pain because of the intertwined physical and psychological variables associated with pain.[12] Accordingly, a psychiatrist may address the cognitive and behavioral components of pain while the other team members focus on the physical aspects of pain. The emphasis of these studies shows that somatic dysfunctions of the spine and its associated pain require a holistic and multidisciplinary approach that leads to better patient outcomes.[12]

Review Questions

References

1.
Yoo WG. Effects of thoracic posture correction exercises on scapular position. J Phys Ther Sci. 2018 Mar;30(3):411-412. [PMC free article: PMC5857448] [PubMed: 29581661]
2.
Louw A, Schmidt SG. Chronic pain and the thoracic spine. J Man Manip Ther. 2015 Jul;23(3):162-8. [PMC free article: PMC4534852] [PubMed: 26308707]
3.
Task Force on the Low Back Pain Clinical Practice Guidelines. American Osteopathic Association Guidelines for Osteopathic Manipulative Treatment (OMT) for Patients With Low Back Pain. J Am Osteopath Assoc. 2016 Aug 01;116(8):536-49. [PubMed: 27455103]
4.
Waxenbaum JA, Reddy V, Futterman B. StatPearls [Internet]. StatPearls Publishing; Treasure Island (FL): Aug 1, 2023. Anatomy, Back, Thoracic Vertebrae. [PubMed: 29083651]
5.
Mrozkowiak M, Walicka-Cupryś K, Magoń G. Comparison of Spinal Curvatures in the Sagittal Plane, as Well as Body Height and Mass in Polish Children and Adolescents Examined in the Late 1950s and in the Early 2000s. Med Sci Monit. 2018 Jun 30;24:4489-4500. [PMC free article: PMC6057267] [PubMed: 29959309]
6.
DeSai C, Reddy V, Agarwal A. StatPearls [Internet]. StatPearls Publishing; Treasure Island (FL): Aug 8, 2023. Anatomy, Back, Vertebral Column. [PubMed: 30247844]
7.
Wong CK, Abraham T, Karimi P, Ow-Wing C. Strain counterstrain technique to decrease tender point palpation pain compared to control conditions: a systematic review with meta-analysis. J Bodyw Mov Ther. 2014 Apr;18(2):165-73. [PubMed: 24725782]
8.
Korr IM. Proprioceptors and somatic dysfunction. J Am Osteopath Assoc. 1975 Mar;74(7):638-50. [PubMed: 124754]
9.
Hindle KB, Whitcomb TJ, Briggs WO, Hong J. Proprioceptive Neuromuscular Facilitation (PNF): Its Mechanisms and Effects on Range of Motion and Muscular Function. J Hum Kinet. 2012 Mar;31:105-13. [PMC free article: PMC3588663] [PubMed: 23487249]
10.
Schiowitz S. Facilitated positional release. J Am Osteopath Assoc. 1990 Feb;90(2):145-6, 151-5. [PubMed: 2407698]
11.
Campbell SM, Winkelmann RR, Walkowski S. Osteopathic manipulative treatment: novel application to dermatological disease. J Clin Aesthet Dermatol. 2012 Oct;5(10):24-32. [PMC free article: PMC3486778] [PubMed: 23125887]
12.
Parkin-Smith GF, Amorin-Woods LG, Davies SJ, Losco BE, Adams J. Spinal pain: current understanding, trends, and the future of care. J Pain Res. 2015;8:741-52. [PMC free article: PMC4631429] [PubMed: 26604815]

Disclosure: Adithi Vemuri declares no relevant financial relationships with ineligible companies.

Disclosure: Kiyomi Goto declares no relevant financial relationships with ineligible companies.

Copyright © 2024, StatPearls Publishing LLC.

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Bookshelf ID: NBK560601PMID: 32809436

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