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Organizing biological data
Loss of SMARCB1 evokes targetable epigenetic vulnerabilities in Epithelioid Sarcoma [ATAC-Seq]
Loss of SMARCB1 evokes targetable epigenetic vulnerabilities in Epithelioid Sarcoma [ChIP-Seq]
Transcriptional response of S. oneidensis MR-1 with lipophilic or hydrophilic ES under different anode potential
Preconditioning With Electrical Stimulation Promotes Flap Survival by Improving Blood Supply Via ROS/Nrf2/HO1 Pathway
Gene expression profile at single cell level of human control, MPHOSPH8-deficient and MORC2-deficient cerebral organoids at day 27.
LncRNA NONMMUT104330.1-miR-709-DDX58 axis mediates enoxaparin sodium bone cement to regulate M2 polarization of macrophages and play an anti-inflammatory immunomodulatory role
Jade1 and the HBO1 complex are spatial-selective cofactors of Oct4
Microbiome associated with bivalves
Microplastics affected bivalve microbial communities
Glycine max
RNA-seq Data of Low Phytic Acid Soybean Mutant Lines and Wild-Type Parental Variety Across Different Developmental Stages and Seasons
Pharmacological targeting of P300/CBP Reveals EWS::FLI1-mediated senescence evasion in Ewing sarcoma
Analysis of miRNA expression upon stimulation of LPS activated THP-1 macrophages with Fasciola hepatica Excretory-Secretory Products (Fh-ES)
Single-cell analysis of bidirectional reprogramming between early embryonic states reveals mechanisms of differential lineage plasticities [scRNA-Seq]
Single-cell analysis of bidirectional reprogramming between early embryonic states reveals mechanisms of differential lineage plasticities [scATAC-seq]
ChIP-seq O-GlcNac in mouse ES cells
CutnRun of O-GlcNac in mouse ES cells in low glucose condition at 6 hours, 8 hours, and 1 week
CutnRun of O-GlcNac in mouse ES cells in high glucose condition
Circadian Clock Regulation in Soybean Senescence: A Transcriptome Analysis of Early and Late Senescence Types
Chip-Seq Raw data in LSD1 WT and KO mouse ES cells
LSD1, H3K4me1 and DNMT1 binding in LSD1 WT and KO mouse ES cells
AtlantECO Consortium jointly owned results
Helminth protein enhances wound healing by inhibiting fibrosis and promoting tissue regeneration
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