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Organizing biological data
Next-Generation Sequencing Facilitates Quantitative Analysis of Control and BET bromodomain Inhibitor-treated Uterine Leiomyosarcoma Cell Transcriptome
Hyperglycemia-triggered lipid peroxidation destabilizes STAT4 and impairs anti-viral Th1 responses in type 2 diabetes
Catalytic-independent functions of INTAC confer sensitivity to BET inhibition [CUT&Tag]
Catalytic-independent functions of INTAC confer sensitivity to BET inhibition [RNA-seq]
Catalytic-independent functions of INTAC confer sensitivity to BET inhibition
CRISPR/Cas9 and cDNA overexpression screens to identify transmembrane transporters involved in PROTAC sensitivity/resistance
Toggling of BRD4 functions is triggered through its phosphorylation by JNK [RNAseq-JQ1]
Transcriptional shut-off of MAP kinase signaling enables pluripotency maintenance during diapause [ChIP-Seq]
BET inhibition reforms the immune microenvironment and alleviates T-cell dysfunction in chronic lymphocytic leukemia
Baricitinib with cyclosporine eliminates acute graft rejection in fully mismatched skin and heart transplant models
Long Terminal Repeats of Gammaretroviruses Retain Stable Expression After Integration Retargeting or Knock-In into the Restrictive Chromatin of Lamina-Associated Domains
Suppressing Aberrant Hedgehog Pathway and Overcoming Resistance to Smoothened Antagonists via Targeting Super-enhancer-driven Transcriptional Dependencies [RNA-seq]
Suppressing Aberrant Hedgehog Pathway and Overcoming Resistance to Smoothened Antagonists via Targeting Super-enhancer-driven Transcriptional Dependencies [ChIP-seq]
Plasticity and lineage commitment of individual Th1 cells are determined by stable T-bet expression quantities
Plasticity and lineage commitment of individual Th1 cells are determined by stable T-bet expression quantities (ChIP-Seq)
Gene expression dynamics of branching morphogenesis in pancreatic cancer organoids (2)
An end-to-end workflow to study newly synthesized mRNA following rapid protein depletion in Saccharomyces cerevisiae
Liver cancer development driven by the AP-1/c-Jun~Fra2 dimer through c-Myc
BET Bromodomain Degradation Disrupts Function but Not 3D Formation of RNA Pol2 Clusters
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