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Accession: PRJNA474525 ID: 474525

Coronary artery disease genes SMAD3 and TCF21 promote opposing interactive genetic programs that regulate smooth muscle cell differentiation and disease risk

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series
AccessionPRJNA474525; GEO: GSE115319
TypeUmbrella project
SubmissionRegistration date: 4-Jun-2018
Quertermous, Medicine Cardiology, Stanford University
RelevanceSuperseries
Project Data:
Resource NameNumber
of Links
Sequence data
SRA Experiments8
Publications
PubMed1
PMC1
Other datasets
BioSample8
GEO DataSets3
GEO Data Details
ParameterValue
Data volume, Supplementary Mbytes579
SRA Data Details
ParameterValue
Data volume, Gbases57
Data volume, Mbytes23088
Coronary artery disease genes SMAD3 and TCF21 promote opposing interactive genetic programs that regulate smooth muscle cell differentiation and disease risk encompasses the following 2 sub-projects:
Project TypeNumber of Projects
Epigenomics1
BioProject
accession
OrganismTitle
PRJNA474527Homo sapiensCoronary artery disease genes SMAD3 and TCF21 promote opposing interactive genetic programs that regulate smooth muscle cell differentiation and disease risk [ChIP-seq] (Quertermous, Medicine Cardiology,...)
Transcriptome or Gene expression1
BioProject
accession
OrganismTitle
PRJNA474526Homo sapiensCoronary artery disease genes SMAD3 and TCF21 promote opposing interactive genetic programs that regulate smooth muscle cell differentiation and disease risk [RNA-seq] (Quertermous, Medicine Cardiology,...)

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