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Accession: PRJNA345198 ID: 345198

Cockayne syndrome A and B proteins regulate the transcription arrest upon genotoxic stress through a ubiquitin/proteasome degradation process

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series
AccessionPRJNA345198; GEO: GSE87564
TypeUmbrella project
PublicationsEpanchintsev A et al., "Cockayne's Syndrome A and B Proteins Regulate Transcription Arrest after Genotoxic Stress by Promoting ATF3 Degradation.", Mol Cell, 2017 Dec 21;68(6):1054-1066.e6
SubmissionRegistration date: 3-Oct-2016
IGBMC (CNRS/INSERM/UDS)
RelevanceSuperseries
Project Data:
Resource NameNumber
of Links
Sequence data
SRA Experiments25
Publications
PubMed1
Other datasets
BioSample25
GEO DataSets3
GEO Data Details
ParameterValue
Data volume, Supplementary Mbytes29
SRA Data Details
ParameterValue
Data volume, Gbases108
Data volume, Mbytes37953
Cockayne syndrome A and B proteins regulate the transcription arrest upon genotoxic stress through a ubiquitin/proteasome degradation process encompasses the following 2 sub-projects:
Project TypeNumber of Projects
Epigenomics1
BioProject
accession
OrganismTitle
PRJNA345269Homo sapiensCockayne syndrome A and B proteins regulate the transcription arrest upon genotoxic stress through a ubiquitin/proteasome degradation process (ChIP-seq) (IGBMC (CNRS/INSERM/UDS))
Transcriptome or Gene expression1
BioProject
accession
OrganismTitle
PRJNA345197Homo sapiensCockayne syndrome A and B proteins regulate the transcription arrest upon genotoxic stress through a ubiquitin/proteasome degradation process (RNA-seq) (IGBMC (CNRS/INSERM/UDS))

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