This SuperSeries is composed of the SubSeries listed below.
Overall design: Refer to individual Series
| Accession | PRJNA345198; GEO: GSE87564 |
| Type | Umbrella project |
| Publications | Epanchintsev A et al., "Cockayne's Syndrome A and B Proteins Regulate Transcription Arrest after Genotoxic Stress by Promoting ATF3 Degradation.", Mol Cell, 2017 Dec 21;68(6):1054-1066.e6 |
| Submission | Registration date: 3-Oct-2016 IGBMC (CNRS/INSERM/UDS) |
| Relevance | Superseries |
Project Data:
| Resource Name | Number of Links |
|---|
| Sequence data |
| SRA Experiments | 25 |
| Publications |
| PubMed | 1 |
| Other datasets |
| BioSample | 25 |
| GEO DataSets | 3 |
Cockayne syndrome A and B proteins regulate the transcription arrest upon genotoxic stress through a ubiquitin/proteasome degradation process encompasses the following 2 sub-projects:
| Project Type | Number of Projects |
| Epigenomics | 1 |
BioProject accession | Organism | Title |
|---|
| PRJNA345269 | Homo sapiens | Cockayne syndrome A and B proteins regulate the transcription arrest upon genotoxic stress through a ubiquitin/proteasome degradation process (ChIP-seq) (IGBMC (CNRS/INSERM/UDS)) |
|
| Transcriptome or Gene expression | 1 |
BioProject accession | Organism | Title |
|---|
| PRJNA345197 | Homo sapiens | Cockayne syndrome A and B proteins regulate the transcription arrest upon genotoxic stress through a ubiquitin/proteasome degradation process (RNA-seq) (IGBMC (CNRS/INSERM/UDS)) |
|