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Accession: PRJNA344679 ID: 344679

The destiny of the resistance/susceptibility against GCRV has been congenitally destined by epigenetic mechanism in CIK cells

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series
AccessionPRJNA344679; GEO: GSE87414
TypeUmbrella project
PublicationsShang X et al., "The destiny of the resistance/susceptibility against GCRV is controlled by epigenetic mechanisms in CIK cells.", Sci Rep, 2017 Jul 3;7(1):4551
SubmissionRegistration date: 27-Sep-2016
Jianguo Su, College of Fisheries, Huazhong Agricultural University
RelevanceSuperseries
Project Data:
Resource NameNumber
of Links
Sequence data
SRA Experiments9
Publications
PubMed1
PMC1
Other datasets
BioSample9
GEO DataSets4
GEO Data Details
ParameterValue
Data volume, Supplementary Mbytes31
SRA Data Details
ParameterValue
Data volume, Gbases40
Data volume, Mbytes16833
The destiny of the resistance/susceptibility against GCRV has been congenitally destined by epigenetic mechanism in CIK cells encompasses the following 3 sub-projects:
Project TypeNumber of Projects
Epigenomics1
BioProject
accession
OrganismTitle
PRJNA344672Ctenopharyngodon idellaThe destiny of the resistance/susceptibility against GCRV has been congenitally destined by epigenetic mechanism in CIK cells (MeDIP-seq) (Jianguo Su, College of Fisheries,...)
Transcriptome or Gene expression2
BioProject
accession
OrganismTitle
PRJNA344706Ctenopharyngodon idellaThe destiny of the resistance/susceptibility against GCRV has been congenitally destined by epigenetic mechanism in CIK cells [miRNA-seq] (Jianguo Su, College of Fisheries,...)
PRJNA344676Ctenopharyngodon idellaThe destiny of the resistance/susceptibility against GCRV has been congenitally destined by epigenetic mechanism in CIK cells [RNA-seq] (Jianguo Su, College of Fisheries,...)

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