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Accession: PRJNA339061 ID: 339061

Cellular model of colon cancer progression reveals signatures of mRNAs, miRNA, lncRNAs and epigenetic modifications associated with metastasis.

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series
AccessionPRJNA339061; GEO: GSE85688
TypeUmbrella project
PublicationsRokavec M et al., "Cellular Model of Colon Cancer Progression Reveals Signatures of mRNAs, miRNA, lncRNAs, and Epigenetic Modifications Associated with Metastasis.", Cancer Res, 2017 Apr 15;77(8):1854-1867
SubmissionRegistration date: 16-Aug-2016
Hermeking, Institute of Pathology, Ludwig-Maximilians-University Munich
RelevanceSuperseries
Project Data:
Resource NameNumber
of Links
Sequence data
SRA Experiments16
Publications
PubMed1
Other datasets
BioSample16
GEO DataSets5
GEO Data Details
ParameterValue
Data volume, Spots2001248
Data volume, Processed Mbytes59
Data volume, Supplementary Mbytes73
SRA Data Details
ParameterValue
Data volume, Gbases28
Data volume, Mbytes16402
Cellular model of colon cancer progression reveals signatures of mRNAs, miRNA, lncRNAs and epigenetic modifications associated with metastasis. encompasses the following 4 sub-projects:
Project TypeNumber of Projects
Epigenomics2
BioProject
accession
OrganismTitle
PRJNA338751Homo sapiensDNA methylation profiling of four DLD1 colorectal cancer cell derivatives that recapitulate EMT/MET transitions during metastasis. (Hermeking, Institute of Pathology,...)
PRJNA339293Homo sapiensCellular model of colon cancer progression reveals signatures of mRNAs, miRNA, lncRNAs and epigenetic modifications associated with metastasis. (Hermeking, Institute of Pathology,...)
Transcriptome or Gene expression2
BioProject
accession
OrganismTitle
PRJNA339109Homo sapiensmicroRNA profiling of four DLD1 colorectal cancer cell derivatives that recapitulate EMT/MET transitions during metastasis. (Hermeking, Institute of Pathology,...)
PRJNA339111Homo sapiensRNA profiling of four DLD1 colorectal cancer cell derivatives that recapitulate EMT/MET transitions during metastasis. (Hermeking, Institute of Pathology,...)

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