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Accession: PRJNA292649 ID: 292649

Genome-wide profiling of inflammatory cistrome reveals AP-1/c-Jun as a key regulator of TNFalpha-mediated triple-negative breast cancer progression.

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series
AccessionPRJNA292649; GEO: GSE71977
TypeUmbrella project
PublicationsQiao Y et al., "AP-1 Is a Key Regulator of Proinflammatory Cytokine TNFα-mediated Triple-negative Breast Cancer Progression.", J Biol Chem, 2016 Mar 4;291(10):5068-79
SubmissionRegistration date: 12-Aug-2015
Medicinaren 25/Neo, Department of Biosciences and Nutrition, Karolinska Institutet
RelevanceSuperseries
Project Data:
Resource NameNumber
of Links
Sequence data
SRA Experiments4
Publications
PubMed1
PMC1
Other datasets
BioSample4
GEO DataSets3
GEO Data Details
ParameterValue
Data volume, Spots399564
Data volume, Processed Mbytes6
Data volume, Supplementary Mbytes58
SRA Data Details
ParameterValue
Data volume, Gbases9
Data volume, Mbytes5326
Genome-wide profiling of inflammatory cistrome reveals AP-1/c-Jun as a key regulator of TNFalpha-mediated triple-negative breast cancer progression. encompasses the following 2 sub-projects:
Project TypeNumber of Projects
Epigenomics1
BioProject
accession
OrganismTitle
PRJNA292652Homo sapiensGenome-wide profiling of inflammatory cistrome reveals AP-1/c-Jun as a key regulator of TNFalpha-mediated triple-negative breast cancer progression [ChIP-seq] (Medicinaren 25/Neo, Department...)
Transcriptome or Gene expression1
BioProject
accession
OrganismTitle
PRJNA292443Homo sapiensGenome-wide profiling of inflammatory cistrome reveals AP-1/c-Jun as a key regulator of TNFalpha-mediated triple-negative breast cancer progression [microarray] (Department of Biosciences...)

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    Genome-wide profiling of inflammatory cistrome reveals AP-1/c-Jun as a key regulator of TNFalpha-mediated triple-negative breast cancer progression.
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