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Accession: PRJNA160951 ID: 160951

PPARγ is a major driver of the accumulation and phenotype of adipose-tissue Treg cells

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series
AccessionPRJNA160951; GEO: GSE37535
TypeUmbrella project
Publications
  • Cipolletta D et al., "Appearance and disappearance of the mRNA signature characteristic of Treg cells in visceral adipose tissue: age, diet, and PPARγ effects.", Proc Natl Acad Sci U S A, 2015 Jan 13;112(2):482-7
  • Cipolletta D et al., "PPAR-γ is a major driver of the accumulation and phenotype of adipose tissue Treg cells.", Nature, 2012 Jun 28;486(7404):549-53
SubmissionRegistration date: 4-May-2012
CBDM, Microbiology and Immunobiology, Harvard Medical School
RelevanceSuperseries
Project Data:
Resource NameNumber
of Links
Publications
PubMed2
PMC2
Other datasets
GEO DataSets4
GEO Data Details
ParameterValue
Data volume, Spots1358928
Data volume, Processed Mbytes25
Data volume, Supplementary Mbytes225
This project encompasses the following 3 sub-projects:
Project TypeNumber of Projects
Transcriptome or Gene expression3
BioProject
accession
OrganismTitle
PRJNA161103Mus musculusGene expression profile of regulatory T cells (Tregs) isolated from visceral adipose tissue and lymph nodes of mice sufficient and deficient of Pparg expression in Tregs (CBDM, Microbiology and Immunobiology,...)
PRJNA161105Mus musculusExpression data of Pioglitazone- or vehicle-treated CD4+FoxP3- T cells transduced with Foxp3+/- Pparg1 (or Pparg2) (CBDM, Microbiology and Immunobiology,...)
PRJNA161107Mus musculusExpression data of Pioglitazone-, Rosiglitazone-, GW1929- and vehicle-treated CD4+FoxP3- T cells transduced with Foxp3+Pparg1 (CBDM, Microbiology and Immunobiology,...)

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