Homo sapiens (ID 155277) - BioProject

Format

Choose Destination

Accession: PRJNA155277 ID: 155277

Homo sapiens (human)

See Genome Information for Homo sapiens

Navigate Up

Navigate Across

2 additional projects are components of the Cell-type independent MYC target genes reveal a primordial signature involved in biomass accumulation.

Myc plays important roles in cancer and stem cell biology including reprogramming adult cells to pluripotent stem cells. More...

Accession	PRJNA155277; GEO: GSE31964
Data Type	Epigenomics
Scope	Multiisolate
Organism	Homo sapiens[Taxonomy ID: 9606] Eukaryota; Metazoa; Chordata; Craniata; Vertebrata; Euteleostomi; Mammalia; Eutheria; Euarchontoglires; Primates; Haplorrhini; Catarrhini; Hominidae; Homo; Homo sapiens
Publications	Ji H et al., "Cell-type independent MYC target genes reveal a primordial signature involved in biomass accumulation.", PLoS One, 2011;6(10):e26057
Submission	Registration date: 7-Sep-2011 Department of Biostatistics, Johns Hopkins Bloomberg School of Public Health
Relevance	Medical

Project Data:

Parameter	Value
Data volume, Supplementary Mbytes	388

Supplemental Content

Study of Dombrock alleles
Study of Dombrock alleles
Study of Dombrock allele distribution in African and African descent individuals. Identification of new Dombrock allele

BioProject
Homo sapiens
Homo sapiens
MYC binding in human B cells (P493-6) and ESC (H9)

BioProject
Single-cell DNA and RNA sequencing over a 29 year period of a CLL patient demons...
Single-cell DNA and RNA sequencing over a 29 year period of a CLL patient demonstrating evolution of multiple cell clones
Single-cell DNA and RNA sequencing over a 29 year period of a CLL patient demonstrating evolution of multiple cell clones

BioProject
Reduced insulin/IGF-1-like signaling restores germ cell immortality in Caenorhab...
Reduced insulin/IGF-1-like signaling restores germ cell immortality in Caenorhabditis elegans Piwi mutants
Reduced insulin/IGF-1-like signaling restores germ cell immortality in Caenorhabditis elegans Piwi mutants

BioProject
Failure to maternally reprogram histone methylation causes developmental delay d...
Failure to maternally reprogram histone methylation causes developmental delay due to germline transcription in somatic tissues
Failure to maternally reprogram histone methylation causes developmental delay due to germline transcription in somatic tissues

BioProject

Your browsing activity is empty.

Activity recording is turned off.