Similar articles for PMID: 16858412

Year	Number of Results
1995	3
1996	1
1997	4
1998	3
1999	11
2000	9
2001	13
2002	11
2003	11
2004	8
2005	8
2006	7
2007	10
2008	2
2014	1
2017	1
2024	0

1

Tumor suppressor gene identification using retroviral insertional mutagenesis in Blm-deficient mice.

Suzuki T, Minehata K, Akagi K, Jenkins NA, Copeland NG. Suzuki T, et al. EMBO J. 2006 Jul 26;25(14):3422-31. doi: 10.1038/sj.emboj.7601215. Epub 2006 Jul 6. EMBO J. 2006. PMID: 16858412 Free PMC article.

2

Enhanced tumor formation in mice heterozygous for Blm mutation.

Goss KH, Risinger MA, Kordich JJ, Sanz MM, Straughen JE, Slovek LE, Capobianco AJ, German J, Boivin GP, Groden J. Goss KH, et al. Science. 2002 Sep 20;297(5589):2051-3. doi: 10.1126/science.1074340. Science. 2002. PMID: 12242442

3

Mismatch repair genes identified using genetic screens in Blm-deficient embryonic stem cells.

Guo G, Wang W, Bradley A. Guo G, et al. Nature. 2004 Jun 24;429(6994):891-5. doi: 10.1038/nature02653. Nature. 2004. PMID: 15215866 Free article.

4

RecQ helicases: caretakers of the genome.

Hickson ID. Hickson ID. Nat Rev Cancer. 2003 Mar;3(3):169-78. doi: 10.1038/nrc1012. Nat Rev Cancer. 2003. PMID: 12612652 Review.

5

Werner and Bloom helicases are involved in DNA repair in a complementary fashion.

Imamura O, Fujita K, Itoh C, Takeda S, Furuichi Y, Matsumoto T. Imamura O, et al. Oncogene. 2002 Jan 31;21(6):954-63. doi: 10.1038/sj.onc.1205143. Oncogene. 2002. PMID: 11840341

6

Genome-wide phenotype analysis in ES cells by regulated disruption of Bloom's syndrome gene.

Yusa K, Horie K, Kondoh G, Kouno M, Maeda Y, Kinoshita T, Takeda J. Yusa K, et al. Nature. 2004 Jun 24;429(6994):896-9. doi: 10.1038/nature02646. Nature. 2004. PMID: 15215867 Free article.

7

Clinical features of Bloom syndrome and function of the causative gene, BLM helicase.

Kaneko H, Kondo N. Kaneko H, et al. Expert Rev Mol Diagn. 2004 May;4(3):393-401. doi: 10.1586/14737159.4.3.393. Expert Rev Mol Diagn. 2004. PMID: 15137905 Review.

8

Premature aging and predisposition to cancers caused by mutations in RecQ family helicases.

Furuichi Y. Furuichi Y. Ann N Y Acad Sci. 2001 Apr;928:121-31. doi: 10.1111/j.1749-6632.2001.tb05642.x. Ann N Y Acad Sci. 2001. PMID: 11795503 Review.

9

Recql5 and Blm RecQ DNA helicases have nonredundant roles in suppressing crossovers.

Hu Y, Lu X, Barnes E, Yan M, Lou H, Luo G. Hu Y, et al. Mol Cell Biol. 2005 May;25(9):3431-42. doi: 10.1128/MCB.25.9.3431-3442.2005. Mol Cell Biol. 2005. PMID: 15831450 Free PMC article.

10

Structural basis of Bloom syndrome (BS) causing mutations in the BLM helicase domain.

Rong SB, Väliaho J, Vihinen M. Rong SB, et al. Mol Med. 2000 Mar;6(3):155-64. Mol Med. 2000. PMID: 10965492 Free PMC article.

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