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    Results: 4

    1.

    Hepatic VLDL production in ob/ob mice is not stimulated by massive de novo lipogenesis but is less sensitive to the suppressive effects of insulin.

    Wiegman CH, Bandsma RH, Ouwens M, van der Sluijs FH, Havinga R, Boer T, Reijngoud DJ, Romijn JA, Kuipers F.

    Diabetes. 2003 May;52(5):1081-9.PMID: 12716736 [PubMed - indexed for MEDLINE]Related articlesFree article

    2.

    Acute inhibition of glucose-6-phosphate translocator activity leads to increased de novo lipogenesis and development of hepatic steatosis without affecting VLDL production in rats.

    Bandsma RH, Wiegman CH, Herling AW, Burger HJ, ter Harmsel A, Meijer AJ, Romijn JA, Reijngoud DJ, Kuipers F.

    Diabetes. 2001 Nov;50(11):2591-7.PMID: 11679439 [PubMed - indexed for MEDLINE]Related articlesFree article

    3.

    Acute inhibition of hepatic glucose-6-phosphatase does not affect gluconeogenesis but directs gluconeogenic flux toward glycogen in fasted rats. A pharmacological study with the chlorogenic acid derivative S4048.

    van Dijk TH, van der Sluijs FH, Wiegman CH, Baller JF, Gustafson LA, Burger HJ, Herling AW, Kuipers F, Meijer AJ, Reijngoud DJ.

    J Biol Chem. 2001 Jul 13;276(28):25727-35. Epub 2001 May 9.PMID: 11346646 [PubMed - indexed for MEDLINE]Related articlesFree article

    4.

    Excess portal venous long-chain fatty acids induce syndrome X via HPA axis and sympathetic activation.

    Benthem L, Keizer K, Wiegman CH, de Boer SF, Strubbe JH, Steffens AB, Kuipers F, Scheurink AJ.

    Am J Physiol Endocrinol Metab. 2000 Dec;279(6):E1286-93.PMID: 11093916 [PubMed - indexed for MEDLINE]Related articlesFree article

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