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Suppression of signal transducers and activators of transcription 1 (STAT1) in hepatocellular carcinoma is associated with tumor progression.
Hosui A, Klover P, Tatsumi T, Uemura A, Nagano H, Doki Y, Mori M, Hiramatsu N, Kanto T, Hennighausen L, Hayashi N, Takehara T.
Int J Cancer. 2012 Apr 5. doi: 10.1002/ijc.27580. [Epub ahead of print]
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Direct activation of STAT5 by ETV6-LYN fusion protein promotes induction of myeloproliferative neoplasm with myelofibrosis.
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MMTV-Cre transgenes can adversely affect lactation: considerations for conditional gene deletion in mammary tissue.
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The transcription factors signal transducer and activator of transcription 5A (STAT5A) and STAT5B negatively regulate cell proliferation through the activation of cyclin-dependent kinase inhibitor 2b (Cdkn2b) and Cdkn1a expression.
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The gene encoding the hematopoietic stem cell regulator CCN3/NOV is under direct cytokine control through the transcription factors STAT5A/B.
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Stat5 is indispensable for the maintenance of bcr/abl-positive leukaemia.
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Signaling by intrathymic cytokines, not T cell antigen receptors, specifies CD8 lineage choice and promotes the differentiation of cytotoxic-lineage T cells.
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BH3-only protein bid participates in the Bcl-2 network in healthy liver cells.
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The antiapoptotic protein Bcl-xL negatively regulates the bone-resorbing activity of osteoclasts in mice.
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Mcl-1 and Bcl-xL cooperatively maintain integrity of hepatocytes in developing and adult murine liver.
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