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    Results: 1 to 20 of 101

    1.

    T-cell promiscuity in autoimmune diabetes.

    Li L, Wang B, Frelinger JA, Tisch R.

    Diabetes. 2008 Aug;57(8):2099-106. Epub 2008 May 20.PMID: 18492786 [PubMed - indexed for MEDLINE]Related articlesFree article

    2.

    Regulatory and effector CD4 T cells in nonobese diabetic mice recognize overlapping determinants on glutamic acid decarboxylase and use distinct V beta genes.

    Quinn A, McInerney B, Reich EP, Kim O, Jensen KP, Sercarz EE.

    J Immunol. 2001 Mar 1;166(5):2982-91.PMID: 11207247 [PubMed - indexed for MEDLINE]Related articlesFree article

    3.

    Peripheral T cell clones from NOD mice specific for GAD65 peptides: lack of islet responsiveness or diabetogenicity.

    Schloot NC, Daniel D, Norbury-Glaser M, Wegmann DR.

    J Autoimmun. 1996 Jun;9(3):357-63.PMID: 8816971 [PubMed - indexed for MEDLINE]Related articles

    4.

    CD4(+) T cells from glutamic acid decarboxylase (GAD)65-specific T cell receptor transgenic mice are not diabetogenic and can delay diabetes transfer.

    Tarbell KV, Lee M, Ranheim E, Chao CC, Sanna M, Kim SK, Dickie P, Teyton L, Davis M, McDevitt H.

    J Exp Med. 2002 Aug 19;196(4):481-92.PMID: 12186840 [PubMed - indexed for MEDLINE]Related articlesFree article

    5.

    Epitope specificity, cytokine production profile and diabetogenic activity of insulin-specific T cell clones isolated from NOD mice.

    Daniel D, Gill RG, Schloot N, Wegmann D.

    Eur J Immunol. 1995 Apr;25(4):1056-62.PMID: 7537670 [PubMed - indexed for MEDLINE]Related articles

    6.

    Major DQ8-restricted T-cell epitopes for human GAD65 mapped using human CD4, DQA1*0301, DQB1*0302 transgenic IA(null) NOD mice.

    Liu J, Purdy LE, Rabinovitch S, Jevnikar AM, Elliott JF.

    Diabetes. 1999 Mar;48(3):469-77.PMID: 10078545 [PubMed - indexed for MEDLINE]Related articlesFree article

    7.

    On the pathogenicity of autoantigen-specific T-cell receptors.

    Burton AR, Vincent E, Arnold PY, Lennon GP, Smeltzer M, Li CS, Haskins K, Hutton J, Tisch RM, Sercarz EE, Santamaria P, Workman CJ, Vignali DA.

    Diabetes. 2008 May;57(5):1321-30. Epub 2008 Feb 25.PMID: 18299317 [PubMed - indexed for MEDLINE]Related articlesFree article

    8.

    T cells to a dominant epitope of GAD65 express a public CDR3 motif.

    Quinn A, McInerney M, Huffman D, McInerney B, Mayo S, Haskins K, Sercarz E.

    Int Immunol. 2006 Jun;18(6):967-79. Epub 2006 Apr 26.PMID: 16641112 [PubMed - indexed for MEDLINE]Related articlesFree article

    9.

    Prevention of type I diabetes transfer by glutamic acid decarboxylase 65 peptide 206-220-specific T cells.

    Kim SK, Tarbell KV, Sanna M, Vadeboncoeur M, Warganich T, Lee M, Davis M, McDevitt HO.

    Proc Natl Acad Sci U S A. 2004 Sep 28;101(39):14204-9. Epub 2004 Sep 20.PMID: 15381770 [PubMed - indexed for MEDLINE]Related articlesFree article

    10.

    The type and frequency of immunoregulatory CD4+ T-cells govern the efficacy of antigen-specific immunotherapy in nonobese diabetic mice.

    Pop SM, Wong CP, He Q, Wang Y, Wallet MA, Goudy KS, Tisch R.

    Diabetes. 2007 May;56(5):1395-402. Epub 2007 Feb 22.PMID: 17317763 [PubMed - indexed for MEDLINE]Related articlesFree article

    11.

    Establishment of T-cell lines from infiltrated NOD islets by stimulation of specific T-cell receptor V beta segments.

    Maugendre D, Legrand B, Olivi M, Bedossa P, Bach JF, Carnaud C.

    J Autoimmun. 1993 Aug;6(4):423-36.PMID: 8105789 [PubMed - indexed for MEDLINE]Related articles

    12.

    Evidence for a preferential V beta usage by the T cells which adoptively transfer diabetes in NOD mice.

    Edouard P, Thivolet C, Bedossa P, Olivi M, Legrand B, Bendelac A, Bach JF, Carnaud C.

    Eur J Immunol. 1993 Mar;23(3):727-33.PMID: 8095459 [PubMed - indexed for MEDLINE]Related articles

    13.

    T cells with multiple fine specificities are used by non-obese diabetic (NOD) mice in the response to GAD(524-543).

    Quinn A, Sercarz EE.

    J Autoimmun. 1996 Jun;9(3):365-70.PMID: 8816972 [PubMed - indexed for MEDLINE]Related articles

    14.

    Evidence for shared recognition of a peptide ligand by a diverse panel of non-obese diabetic mice-derived, islet-specific, diabetogenic T cell clones.

    Yoshida K, Martin T, Yamamoto K, Dobbs C, Münz C, Kamikawaji N, Nakano N, Rammensee HG, Sasazuki T, Haskins K, Kikutani H.

    Int Immunol. 2002 Dec;14(12):1439-47.PMID: 12456592 [PubMed - indexed for MEDLINE]Related articlesFree article

    15.

    A glutamic acid decarboxylase 65-specific Th2 cell clone immunoregulates autoimmune diabetes in nonobese diabetic mice.

    Tisch R, Wang B, Atkinson MA, Serreze DV, Friedline R.

    J Immunol. 2001 Jun 1;166(11):6925-36.PMID: 11359854 [PubMed - indexed for MEDLINE]Related articlesFree article

    16.

    Quantitative thresholds of MHC class II I-E expressed on hemopoietically derived antigen-presenting cells in transgenic NOD/Lt mice determine level of diabetes resistance and indicate mechanism of protection.

    Hanson MS, Cetkovic-Cvrlje M, Ramiya VK, Atkinson MA, Maclaren NK, Singh B, Elliott JF, Serreze DV, Leiter EH.

    J Immunol. 1996 Aug 1;157(3):1279-87.PMID: 8757636 [PubMed - indexed for MEDLINE]Related articles

    17.

    Immunization with the larger isoform of mouse glutamic acid decarboxylase (GAD67) prevents autoimmune diabetes in NOD mice.

    Elliott JF, Qin HY, Bhatti S, Smith DK, Singh RK, Dillon T, Lauzon J, Singh B.

    Diabetes. 1994 Dec;43(12):1494-9.PMID: 7525393 [PubMed - indexed for MEDLINE]Related articles

    18.

    Spontaneous autoimmune diabetes in monoclonal T cell nonobese diabetic mice.

    Verdaguer J, Schmidt D, Amrani A, Anderson B, Averill N, Santamaria P.

    J Exp Med. 1997 Nov 17;186(10):1663-76.PMID: 9362527 [PubMed - indexed for MEDLINE]Related articlesFree article

    19.

    Peri-islet infiltrates of young non-obese diabetic mice display restricted TCR beta-chain diversity.

    Galley KA, Danska JS.

    J Immunol. 1995 Mar 15;154(6):2969-82.PMID: 7533189 [PubMed - indexed for MEDLINE]Related articles

    20.

    Pancreatic IL-4 expression results in islet-reactive Th2 cells that inhibit diabetogenic lymphocytes in the nonobese diabetic mouse.

    Gallichan WS, Balasa B, Davies JD, Sarvetnick N.

    J Immunol. 1999 Aug 1;163(3):1696-703.PMID: 10415077 [PubMed - indexed for MEDLINE]Related articlesFree article

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