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1. |
Observational study of gene-disease association and gene-environment interaction. (HuGE Navigator) |
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2. |
This analysis revealed significant differences in both allele frequencies and genotype distributions of the STX1A gene in migraine. |
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3. |
Homology with vesicle fusion mediator syntaxin-1a predicts determinants of epimorphin/syntaxin-2 function in mammary epithelial morphogenesis.( |
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4. |
Direct interaction of otoferlin with syntaxin 1A, SNAP-25, and the L-type voltage-gated calcium channel Cav1.3. |
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STX1A might influence the serotonergic system during neurodevelopment |
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6. |
SNARE complex-related genes STX1A, VAMP2 and SNAP25 do not play a major role in susceptibility to schizophrenia in the Japanese population |
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7. |
The expression of the SNARE protein SNAP-25 and its cellular homologue SNAP-23, as well as syntaxin1 and VAMP (vesicle-associated membrane protein) in samples of normal parathyroid tissue, chief cell adenoma, and parathyroid carcinoma, was examined. |
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8. |
A lower frequency of the PRM -352T allele of the STX1A gene in overweight/obese subjects impaires glucose regulation, particularly among individuals with combined glucose intolerance and overt diabetes. |
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9. |
Observational study of gene-disease association. (HuGE Navigator) |
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10. |
We have identified a new three-gene classifier that is independent of and improves on stage to stratify early-stage NSCLC patients with significantly different prognoses. |
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11. |
Norepinephrine transpossrter/syntaxin 1A complex rapidly redistributes, upon amphetamine treatment, when mechanisms supported by the transporter's NH2 terminus are eliminated. |
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12. |
Syntaxin-1A actions on sulfonylurea receptor 2A blocks acidic pH-induced cardiac K(ATP) channel activation |
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13. |
SNAP-25 traffics to the plasma membrane by a syntaxin-independent mechanism. |
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14. |
cleavage of APP but not syntaxin 1 is independent of cell surface regulation by extracellular ligands |
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15. |
A significant genetic association was found between schizophrenia and an intron 7 single nucleotide polymorphism (SNP) tested. Haplotype analysis supported the association with several significant values that appear to be driven by the intron 7 SNP. |
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16. |
interacts with CFTR protein differently from interactions with SNARE. |
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Syn-1A binds both NBFs of SUR1 and SUR2A but appears to exhibit distinct interactions with NBF2 of these SUR proteins in modulating the KATP channels in islet beta cells and cardiac myocytes |
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18. |
Syntaxin/Munc18 interactions in the late events during vesicle fusion and release in exocytosis |
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19. |
Munc18b binds to syntaxins 1A, 2, and 3 and regulates vesicle transport to the apical plasma membrane |
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20. |
the mechanisms involved in Syn1A-K(v) interactions vary significantly between K(v) channels, thus providing a wide scope for Syn1A modulation of exocytosis and membrane excitability |
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21. |
interacts with dopamine transporter |
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22. |
CFTR channels are coordinately regulated by two cognate t-SNAREs, SNAP-23 (synaptosome-associated protein of 23 kDa) and syntaxin 1A. |
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23. |
analysis of the spatially distinct modes of munc18-syntaxin 1 interaction |
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24. |
the H(abc) domain has a role in membrane trafficking and targeting of syntaxin 1A |