Watanabe C, Komoto S, Hokari R, Kurihara C, Okada Y, Hozumi H, Higashiyama M, Sakuraba A, Tomita K, Tsuzuki Y, Kawaguchi A, Nagao S, Ogata S, Miura S.

J Gastroenterol. 2013 Jun 12. [Epub ahead of print]

PMID:: 23754511; [PubMed - as supplied by publisher]

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Influence of malting conditions on sorghum (Sorghum bicolor (L.) Moench) as a raw material for fermented beverages.

Hassani A, Zarnkow M, Becker T.

Food Sci Technol Int. 2013 Jun 10. [Epub ahead of print]

PMID:: 23751551; [PubMed - as supplied by publisher]

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Effect of heat treatment on wheat dough rheology and wheat protein solubility.

Mann J, Schiedt B, Baumann A, Conde-Petit B, Vilgis TA.

Food Sci Technol Int. 2013 Jun 10. [Epub ahead of print]

PMID:: 23751547; [PubMed - as supplied by publisher]

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Inadequate Nutrient Intake in Patients with Celiac Disease: Results from a German Dietary Survey.

Martin J, Geisel T, Maresch C, Krieger K, Stein J.

Digestion. 2013 Jun 6;87(4):240-246. [Epub ahead of print]

PMID:: 23751356; [PubMed - as supplied by publisher]

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Comparative studies by IR, Raman, and surface-enhanced Raman spectroscopy of azodicarbonamide, biurea and semicarbazide hydrochloride.

Xie Y, Li P, Zhang J, Wang H, Qian H, Yao W.

Spectrochim Acta A Mol Biomol Spectrosc. 2013 May 25;114C:80-84. doi: 10.1016/j.saa.2013.05.055. [Epub ahead of print]

PMID:: 23751223; [PubMed - as supplied by publisher]

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Short communication: Supplementing lysine and methionine in a lactation diet containing a high concentration of wet corn gluten feed did not alter milk protein yield.

Mullins CR, Weber D, Block E, Smith JF, Brouk MJ, Bradford BJ.

J Dairy Sci. 2013 Jun 5. doi:pii: S0022-0302(13)00412-8. 10.3168/jds.2013-6755. [Epub ahead of print]

PMID:: 23746581; [PubMed - as supplied by publisher]

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Discordant patterns of bacterial translocation markers and implications for innate immune imbalances in schizophrenia.

Severance EG, Gressitt KL, Stallings CR, Origoni AE, Khushalani S, Leweke FM, Dickerson FB, Yolken RH.

Schizophr Res. 2013 Jun 6. doi:pii: S0920-9964(13)00273-9. 10.1016/j.schres.2013.05.018. [Epub ahead of print]

PMID:: 23746484; [PubMed - as supplied by publisher]

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Use of whole grain and refined flour from tannin and non-tannin sorghum (Sorghum bicolor (L.) Moench) varieties in frybread.

Rose DJ, Williams E, Mkandawire NL, Weller CL, Jackson DS.

Food Sci Technol Int. 2013 Jun 6. [Epub ahead of print]

PMID:: 23744121; [PubMed - as supplied by publisher]

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Men with celiac disease are shorter than their peers in the general population.

Sonti R, Lebwohl B, Lewis SK, Abu Daya H, Klavan H, Aguilar K, Green PH.

Eur J Gastroenterol Hepatol. 2013 Jun 5. [Epub ahead of print]

PMID:: 23743561; [PubMed - as supplied by publisher]

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Identification of a Hepatoprotective Peptide in Wheat Gluten Hydrolysate against D-Galactosamine-Induced Acute Hepatitis in Rat.

Sato K, Egashira Y, Ono S, Mochizuki S, Shimmura Y, Suzuki Y, Nagata M, Hashimoto K, Kiyono T, Park EY, Nakamura Y, Itabashi M, Sakata Y, Furuta S, Sanada H.

J Agric Food Chem. 2013 Jun 7. [Epub ahead of print]

PMID:: 23742096; [PubMed - as supplied by publisher]

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[Gluten tolerance as a result of earlier exposure?].

Vriezinga SL, Mearin ML.

Ned Tijdschr Geneeskd. 2013;157(23):A6349. Dutch.

PMID:: 23739606; [PubMed - in process]

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Collagenous sprue: Don´t forget connective tissue in chronic diarrhea evaluation.

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Rev Esp Enferm Dig. 2013 May;105(3):171-174.

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Bone mineral density in adult coeliac disease: An updated review.

Lucendo AJ, García-Manzanares A.

Rev Esp Enferm Dig. 2013 May;105(3):154-162.

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CYP3A4-Catalyzed Simvastatin Metabolism as a Non-Invasive Marker of Small Intestinal Health in Celiac Disease.

Morón B, Verma AK, Das P, Taavela J, Dafik L, Diraimondo TR, Albertelli MA, Kraemer T, Mäki M, Khosla C, Rogler G, Makharia GK.

Am J Gastroenterol. 2013 Jun 4. doi: 10.1038/ajg.2013.151. [Epub ahead of print]

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Young adults with coeliac disease may be at increased risk of early atherosclerosis.

De Marchi S, Chiarioni G, Prior M, Arosio E.

Aliment Pharmacol Ther. 2013 Jun 3. doi: 10.1111/apt.12360. [Epub ahead of print]

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The relationship between adiposity and stature in prepubertal children with celiac disease.

Nwosu BU, Snook RI, Maranda L.

J Pediatr Endocrinol Metab. 2013 May 24:1-6. doi: 10.1515/jpem-2012-0312. [Epub ahead of print]

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Figure 6CD associated bacteria suppress the IL-17A response to gluten in children born during the “epidemic”.

IL-17A (a) and IL-10 (b) responses to ex vivo challenge with gluten digest alone (Gluten digest) and the combination of gluten digest and a mixture of CD associated bacteria (Gluten digest+bacteria) shown as fold increase calculated as the ratio over the individual medium control for patients #1 through 8 and grouped according to the IL-17A response to challenge with gluten digest alone. Horizontal bars indicate medians, boxes indicate the 25^th to 75^th IQR, and whiskers indicate the range. Filled boxes and bars: Treated CD patients with strong IL-17A responses to challenge with gluten digest alone, all born during the Swedish CD epidemic. Open boxes and bars: Treated CD patents with weak IL-17A responses to challenge with gluten digest alone, all born after the epidemic. n = 4 in each group.

Intestinal T-cell Responses in Celiac Disease - Impact of Celiac Disease Associated Bacteria

PLoS One. 2013;8(1):e53414.
Figure 3Potential Therapies Targeting Each Stage of Celiac Disease Progression

Immunotoxic gluten peptides may be eliminated at the source through selection or engineering of non-toxic varieties of gluten-containing grains. Exposure to gluten peptides may be avoided by means of dietary abstention. Gluten peptides' proteolytic resistance may be countered via oral enzyme therapy with glutenases. Invasion of gluten peptides across the intestinal epithelium may be prevented by targeting mediators of epithelial permeability, such as proinflammatory cytokines IFN-γ and TNF-α. Pharmacological inhibition of TG2 represents a means of preventing the activation of native gluten peptides to their deamidated forms. Finally, the deleterious response to gluten peptides may be controlled by prophylactic vaccination, by blocking HLA DQ2-mediated presentation of gluten peptides, or by targeting the mediators of gluten-induced damage, such as IL-15 and NKG2D.

Parallels between Pathogens and Gluten Peptides in Celiac Sprue

PLoS Pathog. 2008 February;4(2):e34.
Figure 4

Change in gastrointestinal symptoms as measured by the GSRS and CeD-GSRS during and after gluten challenge in the gluten challenge/placebo drug cohort compared to the aggregated gluten-free control cohorts and the Larazotide-treated gluten challenge groups. Time course of mean change from baseline in the total (a) Gastrointestinal Symptom Rating Scale (GSRS) score and (b) Celiac Disease (CeD-GSRS) scores. The gluten control group includes patients who received placebo drug and the gluten challenge (n=13). Gluten-free control includes patients who received placebo drug or 8 mg larazotide acetate and gluten placebo (n=20). Patients who received larazotide acetate and the gluten challenge were also combined into one group (n=38). P values were calculated using an analysis of covariance model with treatment as a fixed effect and the corresponding baseline value as a covariate.

A Randomized, Double-Blind Study of Larazotide Acetate to Prevent the Activation of Celiac Disease During Gluten Challenge

Am J Gastroenterol. 2012 October;107(10):1554-1562.
Figure 2Antigen Presentation and Production of Antibodies to Gluten Peptides and Tissue Transglutaminase (tTG)

In the subepithelium of the small intestine, native (partially digested) gluten peptides are deamidated by the enzyme tTG. While tTG is ubiquitous, it is predominantly stored intracellularly in an inactive state and released in the presence of inflammation and activated by higher levels of extracellular calcium ions. Deamidation leads to change in shape and charge of the gluten peptides, permitting high-affinity binding to HLA-DQ2 and -DQ8 on APCs such as dendritic cells and macrophages. Only HLA-DQ2 and -DQ8 are able to bind gluten peptides strongly enough to trigger an inflammatory reaction, so the presence of at least 1 of these molecules is a prerequisite for development of celiac disease. Naive T cells that have been activated by deamidated gluten presented by APCs are then able to stimulate both a T_H1 cytotoxic and T_H2 humoral antibody response. The T_H2 response leads to production of antibodies against native gluten peptide, deamidated gluten peptide, and tTG. Antibodies to the self-protein tTG are produced because tTG is often still complexed with deamidated gluten peptides during presentation by APCs. This directed anti-self immune response is the major autoimmune component of celiac disease. TCR indicates T-cell receptor; IFN, interferon.

Celiac Disease Diagnosis and Management

JAMA. 2011 October 12;306(14):1582-1592.

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Remission without insulin therapy on gluten-free diet in a 6-year old boy with type 1 diabetes mellitus. [BMJ Case Rep. 2012]
Remission without insulin therapy on gluten-free diet in a 6-year old boy with type 1 diabetes mellitus.
Sildorf SM, Fredheim S, Svensson J, Buschard K. BMJ Case Rep. 2012 Jun 21; 2012. Epub 2012 Jun 21.
Gluten causes gastrointestinal symptoms in subjects without celiac disease: a double-blind randomized placebo-controlled trial. [Am J Gastroenterol. 2011]
Gluten causes gastrointestinal symptoms in subjects without celiac disease: a double-blind randomized placebo-controlled trial.
Biesiekierski JR, Newnham ED, Irving PM, Barrett JS, Haines M, Doecke JD, Shepherd SJ, Muir JG, Gibson PR. Am J Gastroenterol. 2011 Mar; 106(3):508-14; quiz 515. Epub 2011 Jan 11.
Spectrum of gluten-related disorders: consensus on new nomenclature and classification. [BMC Med. 2012]
Spectrum of gluten-related disorders: consensus on new nomenclature and classification.
Sapone A, Bai JC, Ciacci C, Dolinsek J, Green PH, Hadjivassiliou M, Kaukinen K, Rostami K, Sanders DS, Schumann M, et al. BMC Med. 2012 Feb 7; 10:13. Epub 2012 Feb 7.

1252 free full-text articles in PubMed Central

Update on the diagnosis and management of refractory coeliac disease. [Gastroenterol Res Pract. 2013]
Update on the diagnosis and management of refractory coeliac disease.
Nijeboer P, van Wanrooij RL, Tack GJ, Mulder CJ, Bouma G. Gastroenterol Res Pract. 2013; 2013:518483. Epub 2013 May 9.
Search for atoxic cereals: a single blind, cross-over study on the safety of a single dose of Triticum monococcum, in patients with celiac disease. [BMC Gastroenterol. 2013]
Search for atoxic cereals: a single blind, cross-over study on the safety of a single dose of Triticum monococcum, in patients with celiac disease.
Zanini B, Petroboni B, Not T, Di Toro N, Villanacci V, Lanzarotto F, Pogna N, Ricci C, Lanzini A. BMC Gastroenterol. 2013 May 24; 13(1):92. Epub 2013 May 24.
Differential IL-13 Production by Small Intestinal Leukocytes in Active Coeliac Disease versus Refractory Coeliac Disease. [Mediators Inflamm. 2013]
Differential IL-13 Production by Small Intestinal Leukocytes in Active Coeliac Disease versus Refractory Coeliac Disease.
Gross S, van Wanrooij RL, Nijeboer P, Gelderman KA, Cillessen SA, Meijer GA, Mulder CJ, Bouma G, von Blomberg BM, Bontkes HJ. Mediators Inflamm. 2013; 2013:939047. Epub 2013 Apr 15.

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