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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1961 1
1965 1
1973 1
1975 5
1976 6
1977 2
1978 11
1979 5
1980 10
1981 13
1982 13
1983 16
1984 7
1985 7
1986 10
1987 6
1988 4
1989 7
1990 6
1991 9
1992 6
1993 6
1994 5
1995 4
1996 5
1997 2
1998 4
1999 1
2000 1
2001 5
2002 2
2003 3
2005 2
2006 1
2007 4
2008 2
2009 3
2010 1
2011 3
2012 3
2013 2
2014 2
2016 3
2018 3
2019 1
2020 1
2023 2
2024 0

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216 results

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Page 1
Comprehensive cell atlas of the first-trimester developing human brain.
Braun E, Danan-Gotthold M, Borm LE, Lee KW, Vinsland E, Lönnerberg P, Hu L, Li X, He X, Andrusivová Ž, Lundeberg J, Barker RA, Arenas E, Sundström E, Linnarsson S. Braun E, et al. Science. 2023 Oct 13;382(6667):eadf1226. doi: 10.1126/science.adf1226. Epub 2023 Oct 13. Science. 2023. PMID: 37824650
We described detailed differentiation trajectories of the human forebrain and midbrain and found a large number of region-specific glioblasts that mature into distinct pre-astrocytes and pre-oligodendrocyte precursor cells. ...
We described detailed differentiation trajectories of the human forebrain and midbrain and found a large number of region-specific gliobl …
Origin of microglia.
Kaur C, Hao AJ, Wu CH, Ling EA. Kaur C, et al. Microsc Res Tech. 2001 Jul 1;54(1):2-9. doi: 10.1002/jemt.1114. Microsc Res Tech. 2001. PMID: 11526953 Review.
A second major view has held that microglia are of neuroectodermal origin; the cells either originate from glioblasts or the germinal matrix. Another school of thought is that microglia are derived from blood monocytes. ...
A second major view has held that microglia are of neuroectodermal origin; the cells either originate from glioblasts or the germinal …
Mutations in lottchen cause cell fate transformations in both neuroblast and glioblast lineages in the Drosophila embryonic central nervous system.
Buescher M, Chia W. Buescher M, et al. Development. 1997 Feb;124(3):673-81. doi: 10.1242/dev.124.3.673. Development. 1997. PMID: 9043082
Here we describe the genetic and phenotypic analysis of lottchen (ltt), a novel gene whose loss of function causes a change in the identity of at least one NB as well as cell fate transformations within the lateral glioblast lineage. In wildtype embryos the parental NB of …
Here we describe the genetic and phenotypic analysis of lottchen (ltt), a novel gene whose loss of function causes a change in the identity …
Glioblast migration in the optic stalk of the chick embryo.
Navascués J, Martín-Partido G, Alvarez IS, Rodríguez-Gallardo L, García-Martínez V. Navascués J, et al. Anat Embryol (Berl). 1987;176(1):79-85. doi: 10.1007/BF00309755. Anat Embryol (Berl). 1987. PMID: 3605653
Light and electron microscopy was used to study the chick embryo optic stalk from Hamburger and Hamilton stages 21 to 29. Observations of glioblast morphology in different developmental stages suggest that these cells undergo radial migration toward peripheral regions of t …
Light and electron microscopy was used to study the chick embryo optic stalk from Hamburger and Hamilton stages 21 to 29. Observations of …
Potential improvement of survival statistics for glioblastoma multiforme (WHO IV).
Goldsmith HS. Goldsmith HS. Surg Neurol Int. 2019 Jun 28;10:123. doi: 10.25259/SNI-185-2019. eCollection 2019. Surg Neurol Int. 2019. PMID: 31528459 Free PMC article.
The present-day treatment of a glioblastoma multiforme IV (glioblast) is by surgery, radiation, and chemotherapy. Unfortunately, the current treatment has not significantly improved the survival statistics of this tumor. ...Combining a tumor resection using 5-ALA and placi …
The present-day treatment of a glioblastoma multiforme IV (glioblast) is by surgery, radiation, and chemotherapy. Unfortunately, the …
Inhibition by calmodulin antagonists of glioblast DNA synthesis and morphological changes induced by glia maturation factor.
Okumura K, Kato T, Ito J, Tanaka R. Okumura K, et al. Brain Res. 1982 Apr;255(4):662-7. doi: 10.1016/0165-3806(82)90063-3. Brain Res. 1982. PMID: 7074367
Calmodulin antagonists, N-(6-aminohexyl)-5-chloro-1-naphthalene sulfonamide hydrochlorides (W-7) and trifluoperazine (TFP), markedly inhibited both morphological transforming and mitogenic activities of glia maturation factor (GMF) on rat fetal glioblasts in culture. In th …
Calmodulin antagonists, N-(6-aminohexyl)-5-chloro-1-naphthalene sulfonamide hydrochlorides (W-7) and trifluoperazine (TFP), markedly inhibit …
Cell Calcium Imaging as a Reliable Method to Study Neuron-Glial Circuits.
de Melo Reis RA, Freitas HR, de Mello FG. de Melo Reis RA, et al. Front Neurosci. 2020 Oct 2;14:569361. doi: 10.3389/fnins.2020.569361. eCollection 2020. Front Neurosci. 2020. PMID: 33122991 Free PMC article. Review.
SCCI has also been used as a method to associate phenotypic markers during development and stage transitions in progenitors, stem, vascular cells, neuro- and glioblasts, neurons, astrocytes, oligodendrocytes, and microglia that operate through ion channels, transporters, a …
SCCI has also been used as a method to associate phenotypic markers during development and stage transitions in progenitors, stem, vascular …
Oligosaccharide alterations of rat glioblast membrane-bound glycoproteins during differentiation induced by glia maturation factor.
Ito J, Kato T, Tanaka R. Ito J, et al. Neurochem Int. 1986;8(1):31-40. doi: 10.1016/0197-0186(86)90097-5. Neurochem Int. 1986. PMID: 20493026
Differentiation of normal glioblasts was induced by glia maturation factor (GMF), and the structural change in the oligosaccharide chains of the plasmalemmal glycoproteins was investigated. ...The N-linked oligosaccharides corresponding to isomaltohepta- decaose and larger …
Differentiation of normal glioblasts was induced by glia maturation factor (GMF), and the structural change in the oligosaccharide ch …
216 results