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Page 1
Distribution and biotransformation of methyl mercuric chloride in different tissues of mice.
Mehra M, Choi BH. Mehra M, et al. Acta Pharmacol Toxicol (Copenh). 1981 Jul;49(1):28-37. doi: 10.1111/j.1600-0773.1981.tb00866.x. Acta Pharmacol Toxicol (Copenh). 1981. PMID: 7336962
The distribution of 203Hg radioactivity has been studied in various organs of adult male and female mice from one hour to 21 days after treating with 203 Hg-labeled methyl mercuric chloride (MMC). The amount of methyl mercury (MeHg) and inorganic mercury (Hg) has al …
The distribution of 203Hg radioactivity has been studied in various organs of adult male and female mice from one hour to 21 days after trea …
Kinetics of chemokines and their receptors in mercuric chloride-induced tubulointerstitial lesions in brown Norway rats.
Suzuki K, Kanabayashi T, Nakayama H, Doi K. Suzuki K, et al. Exp Mol Pathol. 2003 Aug;75(1):58-67. doi: 10.1016/s0014-4800(03)00028-5. Exp Mol Pathol. 2003. PMID: 12834626
BN rats were injected subcutaneously with 1 mg/kg b.w. of mercuric chloride one or three times. The kidney was examined histopathologically and the kinetics of chemokines and their receptors in the kidney were also examined using immunohistochemistry a …
BN rats were injected subcutaneously with 1 mg/kg b.w. of mercuric chloride one or three times. The kidney was examined …
Changes in the renal function after acute mercuric chloride exposure in the rat are associated with renal vascular endothelial dysfunction and proximal tubule NHE3 inhibition.
Vieira JVDA, Marques VB, Vieira LV, Crajoinas RO, Shimizu MHM, Seguro AC, Carneiro MTWD, Girardi ACC, Vassallo DV, Dos Santos L. Vieira JVDA, et al. Toxicol Lett. 2021 May 1;341:23-32. doi: 10.1016/j.toxlet.2021.01.014. Epub 2021 Jan 18. Toxicol Lett. 2021. PMID: 33476711 Free article.
Mercury is an environmental pollutant and a threat to human health. Mercuric chloride (HgCl(2))-induced acute renal failure has been described by several reports, but the mechanisms of renal dysfunction remain elusive. ...
Mercury is an environmental pollutant and a threat to human health. Mercuric chloride (HgCl(2))-induced acute renal failure ha …
Mercuric chloride-induced tubulonecrosis in the rat kidney: the recovery phase.
Haagsma BH, Pound AW. Haagsma BH, et al. Br J Exp Pathol. 1980 Jun;61(3):229-41. Br J Exp Pathol. 1980. PMID: 7426379 Free PMC article.
The tubules regenerated from surviving squamoid cells at both ends of the necrotic segments, which often appeared to arise from epithelial cells that had cast off superficial "dead cytoplasm" to leave the basal parts containing the nucleus in a rim of cytoplasm. The tubular ep
The tubules regenerated from surviving squamoid cells at both ends of the necrotic segments, which often appeared to arise from epithelial c …
Toxicology and Carcinogenesis Studies of Mercuric Chloride (CAS No. 7487-94-7) in F344 Rats and B6C3F1 Mice (Gavage Studies).
National Toxicology Program. National Toxicology Program. Natl Toxicol Program Tech Rep Ser. 1993 Feb;408:1-260. Natl Toxicol Program Tech Rep Ser. 1993. PMID: 12621522 Free article.
There was no evidence of carcinogenic activity of mercuric chloride in female B6C3F1 mice receiving 5 or 10 mg/kg. Nonneoplastic lesions associated with exposure to mercuric chloride included increased severity of nephropathy in male rats and male and …
There was no evidence of carcinogenic activity of mercuric chloride in female B6C3F1 mice receiving 5 or 10 mg/kg. Nonneoplast …
Ly-6 in kidney is widely expressed on tubular epithelium and vascular endothelium and is up-regulated by interferon gamma.
Blake PG, Madrenas J, Halloran PF. Blake PG, et al. J Am Soc Nephrol. 1993 Nov;4(5):1140-50. doi: 10.1681/ASN.V451140. J Am Soc Nephrol. 1993. PMID: 8305641
Ly-6 expression was much more extensive in the kidney than in other parenchymal organs. Ly-6A.1/E.2 was extensively expressed on vascular endothelium and on tubular epithelium, particularly in the distal nephron. ...Similar up-regulation of Ly-6 was seen in murine l …
Ly-6 expression was much more extensive in the kidney than in other parenchymal organs. Ly-6A.1/E.2 was extensively expressed on vasc …
Bone marrow cells contribute to tubular epithelium regeneration following acute kidney injury induced by mercuric chloride.
Yadav N, Rao S, Bhowmik DM, Mukhopadhyay A. Yadav N, et al. Indian J Med Res. 2012 Aug;136(2):211-20. Indian J Med Res. 2012. PMID: 22960887 Free PMC article.
The relative contributions of eGFP-expressing BMCs were compared in two different approaches to kidney regeneration in the mercuric chloride (HgCl 2 )-induced mouse model of AKI: induced engraftment and forced engraftment. ...INTERPRETATION & CONCLUSIONS: …
The relative contributions of eGFP-expressing BMCs were compared in two different approaches to kidney regeneration in the mercuri
Activity of glucose-6-phosphatase in regenerating tubular epithelium in rat kidney after necrosis induced with mercuric chloride: a light and electronmicroscopical study.
Böti Z, Kóbor J, Ormos J. Böti Z, et al. Br J Exp Pathol. 1982 Dec;63(6):615-24. Br J Exp Pathol. 1982. PMID: 6295432 Free PMC article.
The activity of glucose-6-phosphatase was investigated in the renal proximal tubules of male albino rats after necrosis induced with mercuric chloride. Between the 3rd and 14th days an active zone could be observed in the inner cortex, which in the control animals w …
The activity of glucose-6-phosphatase was investigated in the renal proximal tubules of male albino rats after necrosis induced with merc
Scanning and transmission EM of rat kidney following low dose mercuric chloride administration.
Siegel FL, Bulger RE. Siegel FL, et al. Beitr Pathol. 1975 Dec;156(4):313-42. doi: 10.1016/s0005-8165(75)80043-6. Beitr Pathol. 1975. PMID: 1220669
The development of necrosis and the subsequent regeneration of the proximal tubular epithelium located in the cortex and the outer medulla of the rat kidney following subcutaneous injection of 2 mg of mercuric chloride per kilogram of body weight was o …
The development of necrosis and the subsequent regeneration of the proximal tubular epithelium located in the cortex and the outer me …
The cellular origin and proliferative status of regenerating renal parenchyma after mercuric chloride damage and erythropoietin treatment.
Yen TH, Alison MR, Cook HT, Jeffery R, Otto WR, Wright NA, Poulsom R. Yen TH, et al. Cell Prolif. 2007 Apr;40(2):143-56. doi: 10.1111/j.1365-2184.2007.00423.x. Cell Prolif. 2007. PMID: 17472723 Free PMC article.
Six weeks later recipient mice were assigned to one of four groups: control, mercuric chloride treated, erythropoietin treated and treated with mercuric chloride plus erythropoietin. RESULTS: Tubular injury scores were high 3 days after mercuric
Six weeks later recipient mice were assigned to one of four groups: control, mercuric chloride treated, erythropoietin treated …
62 results