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Plasminogen activator inhibitor-1 secretion of endothelial cells increases fibrinolytic resistance of an in vitro fibrin clot: evidence for a key role of endothelial cells in thrombolytic resistance.
Blood. 1996 May 15;87(10):4204-13.
Blood. 1996.
PMID: 8639779
Free article.
Time-dependent thrombolytic resistance is a critical problem in thrombolytic therapy for acute myocardial infarction. Platelets have been regarded as the main source of plasminogen activator inhibitor-1 (PAI-1) found in occlusive p …
Time-dependent thrombolytic resistance is a critical problem in thrombolytic therapy for acute myocardial infarction. P …
Plasma TAFI and soluble CD40 ligand do not predict reperfusion following thrombolysis for acute myocardial infarction.
Cruden NL, Graham C, Harding SA, Ludlam CA, Fox KA, Newby DE.
Cruden NL, et al.
Thromb Res. 2006;118(2):189-97. doi: 10.1016/j.thromres.2005.06.014. Epub 2005 Aug 1.
Thromb Res. 2006.
PMID: 16055173
INTRODUCTION: Thrombolytic therapy fails to achieve reperfusion in almost a third of patients with acute myocardial infarction. ...CONCLUSIONS: Systemic plasma TAFI, sCD40L and CRP concentrations do not predict reperfusion in patients receiving thrombolytic …
INTRODUCTION: Thrombolytic therapy fails to achieve reperfusion in almost a third of patients with acute myocardial infarction …
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