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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1945 1
1947 2
1948 3
1949 4
1950 8
1951 8
1952 9
1953 7
1954 10
1955 10
1956 22
1957 18
1958 16
1959 16
1960 18
1961 28
1962 20
1963 45
1964 107
1965 107
1966 152
1967 267
1968 237
1969 326
1970 309
1971 435
1972 387
1973 390
1974 482
1975 361
1976 265
1977 235
1978 220
1979 265
1980 224
1981 224
1982 211
1983 248
1984 360
1985 359
1986 335
1987 339
1988 415
1989 520
1990 595
1991 666
1992 691
1993 788
1994 832
1995 901
1996 1039
1997 1156
1998 1159
1999 1183
2000 1345
2001 1374
2002 1597
2003 2080
2004 2308
2005 2394
2006 2801
2007 2913
2008 2853
2009 2971
2010 3293
2011 3435
2012 3614
2013 3781
2014 3821
2015 4170
2016 3984
2017 4162
2018 4298
2019 4575
2020 5106
2021 5440
2022 4858
2023 4396
2024 1905

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91,210 results

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Page 1
Cardiomyocyte Senescence and Cellular Communications Within Myocardial Microenvironments.
Tang X, Li PH, Chen HZ. Tang X, et al. Front Endocrinol (Lausanne). 2020 May 21;11:280. doi: 10.3389/fendo.2020.00280. eCollection 2020. Front Endocrinol (Lausanne). 2020. PMID: 32508749 Free PMC article. Review.
The senescence of cardiomyocyte is also regulated by the non-myocytes (endothelial cells, fibroblasts, and immune cells) in the local microenvironment. On the other hand, the senescent cardiomyocytes alter their phenotypes and subsequently affect the non-m …
The senescence of cardiomyocyte is also regulated by the non-myocytes (endothelial cells, fibroblasts, and immune cells) in th …
Cardiomyocyte Proliferation for Therapeutic Regeneration.
Leach JP, Martin JF. Leach JP, et al. Curr Cardiol Rep. 2018 Jun 14;20(8):63. doi: 10.1007/s11886-018-1011-x. Curr Cardiol Rep. 2018. PMID: 29904823 Review.
PURPOSE OF REVIEW: Current pharmacologic treatments for cardiovascular disease do not correct the underlying cellular defect, the loss of cardiomyocytes. With recent advancements in cardiac regenerative approaches, the induction of endogenous mature cardiomyocyte …
PURPOSE OF REVIEW: Current pharmacologic treatments for cardiovascular disease do not correct the underlying cellular defect, the loss of …
Adult Cardiomyocyte Cell Cycle Detour: Off-ramp to Quiescent Destinations.
Broughton KM, Sussman MA. Broughton KM, et al. Trends Endocrinol Metab. 2019 Aug;30(8):557-567. doi: 10.1016/j.tem.2019.05.006. Epub 2019 Jun 28. Trends Endocrinol Metab. 2019. PMID: 31262545 Free PMC article. Review.
While some of the struggle is attributable to adult cardiomyocytes themselves that are notoriously post-mitotic, another contributory factor rests with difficulty in definitive tracking of adult cardiomyocyte cell cycle and lack of rigorous measures to track prolife …
While some of the struggle is attributable to adult cardiomyocytes themselves that are notoriously post-mitotic, another contributory …
Chemically Defined Culture and Cardiomyocyte Differentiation of Human Pluripotent Stem Cells.
Burridge PW, Holmström A, Wu JC. Burridge PW, et al. Curr Protoc Hum Genet. 2015 Oct 6;87:21.3.1-21.3.15. doi: 10.1002/0471142905.hg2103s87. Curr Protoc Hum Genet. 2015. PMID: 26439715 Free PMC article.
We provide exact guidelines for cell handling under these conditions, including non-enzymatic EDTA passaging, which have been optimized for subsequent cardiomyocyte differentiation. We describe in depth the latest version of our monolayer chemically defined small molecule …
We provide exact guidelines for cell handling under these conditions, including non-enzymatic EDTA passaging, which have been optimized for …
Understanding cardiomyocyte proliferation: an insight into cell cycle activity.
Ponnusamy M, Li PF, Wang K. Ponnusamy M, et al. Cell Mol Life Sci. 2017 Mar;74(6):1019-1034. doi: 10.1007/s00018-016-2375-y. Epub 2016 Sep 30. Cell Mol Life Sci. 2017. PMID: 27695872 Review.
In the recent years, studies on cardiomyocyte proliferation overturned the traditional belief that adult cardiomyocytes permanently withdraw from the cell cycle activity. ...To achieve this, a deep understanding of the fundamental mechanisms involved in cardiomyo …
In the recent years, studies on cardiomyocyte proliferation overturned the traditional belief that adult cardiomyocytes perman …
Cardiomyocyte renewal in the human heart: insights from the fall-out.
Lázár E, Sadek HA, Bergmann O. Lázár E, et al. Eur Heart J. 2017 Aug 7;38(30):2333-2342. doi: 10.1093/eurheartj/ehx343. Eur Heart J. 2017. PMID: 28810672 Free PMC article. Review.
The capacity of the mammalian heart to regenerate cardiomyocytes has been debated over the last decades. However, limitations in existing techniques to track and identify nascent cardiomyocytes have often led to inconsistent results. ...Accurate estimates of card …
The capacity of the mammalian heart to regenerate cardiomyocytes has been debated over the last decades. However, limitations in exis …
Cardiomyocyte proliferation: remove brakes and push accelerators.
He L, Zhou B. He L, et al. Cell Res. 2017 Aug;27(8):959-960. doi: 10.1038/cr.2017.91. Epub 2017 Jul 14. Cell Res. 2017. PMID: 28707671 Free PMC article. Review.
Adult mammalian hearts cannot repair by themselves after injury due to limited proliferation of cardiomyocytes; removal of cell cycle blocker and/or addition of drugs that boost proliferation of cardiomyocytes provide potential means to cardiac regeneration. …
Adult mammalian hearts cannot repair by themselves after injury due to limited proliferation of cardiomyocytes; removal of cell cycle …
Induced Cardiomyocyte Proliferation: A Promising Approach to Cure Heart Failure.
Salama ABM, Gebreil A, Mohamed TMA, Abouleisa RRE. Salama ABM, et al. Int J Mol Sci. 2021 Jul 19;22(14):7720. doi: 10.3390/ijms22147720. Int J Mol Sci. 2021. PMID: 34299340 Free PMC article. Review.
Unlike some lower vertebrates which can completely regenerate their heart, the human heart is a terminally differentiated organ. Cardiomyocytes lost during cardiac injury and heart failure cannot be replaced due to their limited proliferative capacity. Therefore, …
Unlike some lower vertebrates which can completely regenerate their heart, the human heart is a terminally differentiated organ. Cardiomy …
Cardiomyocyte proliferation in zebrafish and mammals: lessons for human disease.
Matrone G, Tucker CS, Denvir MA. Matrone G, et al. Cell Mol Life Sci. 2017 Apr;74(8):1367-1378. doi: 10.1007/s00018-016-2404-x. Epub 2016 Nov 3. Cell Mol Life Sci. 2017. PMID: 27812722 Free PMC article. Review.
Cardiomyocytes proliferate profusely during early development and for a brief period after birth in mammals. ...We discuss the advantages of the zebrafish as a model of cardiomyocyte proliferation, particularly at the embryonic stage. We also identify a number of ke …
Cardiomyocytes proliferate profusely during early development and for a brief period after birth in mammals. ...We discuss the advant …
Cardiomyocyte Ca2+ dynamics: clinical perspectives.
Aronsen JM, Louch WE, Sjaastad I. Aronsen JM, et al. Scand Cardiovasc J. 2016;50(2):65-77. doi: 10.3109/14017431.2015.1136079. Epub 2016 Feb 7. Scand Cardiovasc J. 2016. PMID: 26729487 Review.
In this review, we summarize the role of local Ca(2+) homeostasis in these processes in healthy cardiac muscle cells, and highlight how mismanaged Ca(2+) handling contributes to the pathophysiology of conditions such as cardiac arrhythmia, ischemic heart disease, …
In this review, we summarize the role of local Ca(2+) homeostasis in these processes in healthy cardiac muscle cells, and highlight h …
91,210 results
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