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Results: 4

1.

SFTPC mutations cause SP-C degradation and aggregate formation without increasing ER stress.

Thurm T, Kaltenborn E, Kern S, Griese M, Zarbock R.

Eur J Clin Invest. 2013 Aug;43(8):791-800. doi: 10.1111/eci.12107. Epub 2013 May 24.

PMID:
23701443
[PubMed - indexed for MEDLINE]
2.

The surfactant protein C mutation A116D alters cellular processing, stress tolerance, surfactant lipid composition, and immune cell activation.

Zarbock R, Woischnik M, Sparr C, Thurm T, Kern S, Kaltenborn E, Hector A, Hartl D, Liebisch G, Schmitz G, Griese M.

BMC Pulm Med. 2012 Mar 29;12:15. doi: 10.1186/1471-2466-12-15.

PMID:
22458263
[PubMed - indexed for MEDLINE]
Free PMC Article
3.

Prevalence and predictors of slow flow in angiographically normal coronary arteries.

Arbel Y, Rind E, Banai S, Halkin A, Berliner S, Herz I, Mashav N, Thurm T, Keren G, Finkelstein A.

Clin Hemorheol Microcirc. 2012;52(1):5-14. doi: 10.3233/CH-2012-1538.

PMID:
22387483
[PubMed - indexed for MEDLINE]
4.

A non-BRICHOS surfactant protein c mutation disrupts epithelial cell function and intercellular signaling.

Woischnik M, Sparr C, Kern S, Thurm T, Hector A, Hartl D, Liebisch G, Mulugeta S, Beers MF, Schmitz G, Griese M.

BMC Cell Biol. 2010 Nov 20;11:88. doi: 10.1186/1471-2121-11-88.

PMID:
21092132
[PubMed - indexed for MEDLINE]
Free PMC Article

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