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Items: 6


Long-acting glucose-dependent insulinotropic polypeptide ameliorates obesity-induced adipose tissue inflammation.

Varol C, Zvibel I, Spektor L, Mantelmacher FD, Vugman M, Thurm T, Khatib M, Elmaliah E, Halpern Z, Fishman S.

J Immunol. 2014 Oct 15;193(8):4002-9. doi: 10.4049/jimmunol.1401149. Epub 2014 Sep 12.


Sortilin deficiency improves the metabolic phenotype and reduces hepatic steatosis of mice subjected to diet-induced obesity.

Rabinowich L, Fishman S, Hubel E, Thurm T, Park WJ, Pewzner-Jung Y, Saroha A, Erez N, Halpern Z, Futerman AH, Zvibel I.

J Hepatol. 2015 Jan;62(1):175-81. doi: 10.1016/j.jhep.2014.08.030. Epub 2014 Aug 27.


SFTPC mutations cause SP-C degradation and aggregate formation without increasing ER stress.

Thurm T, Kaltenborn E, Kern S, Griese M, Zarbock R.

Eur J Clin Invest. 2013 Aug;43(8):791-800. doi: 10.1111/eci.12107. Epub 2013 May 24.


The surfactant protein C mutation A116D alters cellular processing, stress tolerance, surfactant lipid composition, and immune cell activation.

Zarbock R, Woischnik M, Sparr C, Thurm T, Kern S, Kaltenborn E, Hector A, Hartl D, Liebisch G, Schmitz G, Griese M.

BMC Pulm Med. 2012 Mar 29;12:15. doi: 10.1186/1471-2466-12-15.


Prevalence and predictors of slow flow in angiographically normal coronary arteries.

Arbel Y, Rind E, Banai S, Halkin A, Berliner S, Herz I, Mashav N, Thurm T, Keren G, Finkelstein A.

Clin Hemorheol Microcirc. 2012;52(1):5-14. doi: 10.3233/CH-2012-1538.


A non-BRICHOS surfactant protein c mutation disrupts epithelial cell function and intercellular signaling.

Woischnik M, Sparr C, Kern S, Thurm T, Hector A, Hartl D, Liebisch G, Mulugeta S, Beers MF, Schmitz G, Griese M.

BMC Cell Biol. 2010 Nov 20;11:88. doi: 10.1186/1471-2121-11-88.

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