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Items: 12

1.

Conceptual approaches for treatment of phosgene inhalation-induced lung injury.

Holmes WW, Keyser BM, Paradiso DC, Ray R, Andres DK, Benton BJ, Rothwell CC, Hoard-Fruchey HM, Dillman JF, Sciuto AM, Anderson DR.

Toxicol Lett. 2016 Feb 26;244:8-20. doi: 10.1016/j.toxlet.2015.10.010. Epub 2015 Nov 10.

PMID:
26562770
2.

Differentiated NSC-34 cells as an in vitro cell model for VX.

Kanjilal B, Keyser BM, Andres DK, Nealley E, Benton B, Melber AA, Andres JF, Letukas VA, Clark O, Ray R.

Toxicol Mech Methods. 2014 Oct;24(7):488-94. doi: 10.3109/15376516.2014.943442. Epub 2014 Sep 11.

PMID:
25045830
3.

Mustard Gas Inhalation Injury: Therapeutic Strategy.

Keyser BM, Andres DK, Holmes WW, Paradiso D, Appell A, Letukas VA, Benton B, Clark OE, Gao X, Ray P, Anderson DR, Ray R.

Int J Toxicol. 2014 May 6;33(4):271-281. [Epub ahead of print]

PMID:
24801489
4.

Morphological and functional differentiation in BE(2)-M17 human neuroblastoma cells by treatment with Trans-retinoic acid.

Andres D, Keyser BM, Petrali J, Benton B, Hubbard KS, McNutt PM, Ray R.

BMC Neurosci. 2013 Apr 18;14:49. doi: 10.1186/1471-2202-14-49.

5.

Postexposure application of Fas receptor small-interfering RNA to suppress sulfur mustard-induced apoptosis in human airway epithelial cells: implication for a therapeutic approach.

Keyser BM, Andres DK, Nealley E, Holmes WW, Benton B, Paradiso D, Appell A, Carpin C, Anderson DR, Smith WJ, Ray R.

J Pharmacol Exp Ther. 2013 Jan;344(1):308-16. doi: 10.1124/jpet.112.199935. Epub 2012 Nov 5.

6.

Sulfur mustard induces apoptosis in lung epithelial cells via a caspase amplification loop.

Ray R, Simbulan-Rosenthal CM, Keyser BM, Benton B, Anderson D, Holmes W, Trabosh VA, Daher A, Rosenthal DS.

Toxicology. 2010 May 27;271(3):94-9. doi: 10.1016/j.tox.2010.03.008. Epub 2010 Mar 11.

PMID:
20226831
7.

Selective blockade of T-type Ca2+ channels suppresses human breast cancer cell proliferation.

Taylor JT, Huang L, Pottle JE, Liu K, Yang Y, Zeng X, Keyser BM, Agrawal KC, Hansen JB, Li M.

Cancer Lett. 2008 Aug 18;267(1):116-24. doi: 10.1016/j.canlet.2008.03.032. Epub 2008 May 1.

PMID:
18455293
8.

Towards selective antagonists of T-type calcium channels: design, characterization and potential applications of NNC 55-0396.

Li M, Hansen JB, Huang L, Keyser BM, Taylor JT.

Cardiovasc Drug Rev. 2005 Summer;23(2):173-96.

PMID:
16007233
9.

Role of high-voltage-activated calcium channels in glucose-regulated beta-cell calcium homeostasis and insulin release.

Taylor JT, Huang L, Keyser BM, Zhuang H, Clarkson CW, Li M.

Am J Physiol Endocrinol Metab. 2005 Nov;289(5):E900-8. Epub 2005 Jun 14.

10.

T-type Ca2+ channels are involved in high glucose-induced rat neonatal cardiomyocyte proliferation.

Li M, Zhang M, Huang L, Zhou J, Zhuang H, Taylor JT, Keyser BM, Whitehurst RM Jr.

Pediatr Res. 2005 Apr;57(4):550-6. Epub 2005 Feb 4.

PMID:
15695594
11.

NNC 55-0396 [(1S,2S)-2-(2-(N-[(3-benzimidazol-2-yl)propyl]-N-methylamino)ethyl)-6-fluoro-1,2,3,4-tetrahydro-1-isopropyl-2-naphtyl cyclopropanecarboxylate dihydrochloride]: a new selective inhibitor of T-type calcium channels.

Huang L, Keyser BM, Tagmose TM, Hansen JB, Taylor JT, Zhuang H, Zhang M, Ragsdale DS, Li M.

J Pharmacol Exp Ther. 2004 Apr;309(1):193-9. Epub 2004 Jan 12.

12.

[Ca2+]i regulates trafficking of Cav1.3 (alpha1D Ca2+ channel) in insulin-secreting cells.

Huang L, Bhattacharjee A, Taylor JT, Zhang M, Keyser BM, Marrero L, Li M.

Am J Physiol Cell Physiol. 2004 Feb;286(2):C213-21. Epub 2003 Oct 8.

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