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Results: 5

1.

Neonatal-onset multisystem inflammatory disease responsive to interleukin-1beta inhibition.

Goldbach-Mansky R, Dailey NJ, Canna SW, Gelabert A, Jones J, Rubin BI, Kim HJ, Brewer C, Zalewski C, Wiggs E, Hill S, Turner ML, Karp BI, Aksentijevich I, Pucino F, Penzak SR, Haverkamp MH, Stein L, Adams BS, Moore TL, Fuhlbrigge RC, Shaham B, Jarvis JN, O'Neil K, Vehe RK, Beitz LO, Gardner G, Hannan WP, Warren RW, Horn W, Cole JL, Paul SM, Hawkins PN, Pham TH, Snyder C, Wesley RA, Hoffmann SC, Holland SM, Butman JA, Kastner DL.

N Engl J Med. 2006 Aug 10;355(6):581-92.

2.

Activation of Syk tyrosine kinase is required for c-Cbl-mediated ubiquitination of Fcepsilon RI and Syk in RBL cells.

Paolini R, Molfetta R, Beitz LO, Zhang J, Scharenberg AM, Piccoli M, Frati L, Siraganian R, Santoni A.

J Biol Chem. 2002 Oct 4;277(40):36940-7. Epub 2002 Jul 26.

3.

SYK is upstream of phosphoinositide 3-kinase in B cell receptor signaling.

Beitz LO, Fruman DA, Kurosaki T, Cantley LC, Scharenberg AM.

J Biol Chem. 1999 Nov 12;274(46):32662-6.

4.

Phosphatidylinositol-3,4,5-trisphosphate (PtdIns-3,4,5-P3)/Tec kinase-dependent calcium signaling pathway: a target for SHIP-mediated inhibitory signals.

Scharenberg AM, El-Hillal O, Fruman DA, Beitz LO, Li Z, Lin S, Gout I, Cantley LC, Rawlings DJ, Kinet JP.

EMBO J. 1998 Apr 1;17(7):1961-72.

5.

Characterization of Cbl tyrosine phosphorylation and a Cbl-Syk complex in RBL-2H3 cells.

Ota Y, Beitz LO, Scharenberg AM, Donovan JA, Kinet JP, Samelson LE.

J Exp Med. 1996 Nov 1;184(5):1713-23.

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