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Alzheimer disease pathology as a host response.
Castellani RJ, Lee HG, Zhu X, Perry G, Smith MA. Castellani RJ, et al. Among authors: perry g, zhu x, lee hg, smith ma. J Neuropathol Exp Neurol. 2008 Jun;67(6):523-31. doi: 10.1097/NEN.0b013e318177eaf4. J Neuropathol Exp Neurol. 2008. PMID: 18520771 Free PMC article. Review.
Identification of amyloid-beta and tau as the major protein components of senile plaques and neurofibrillary tangles, respectively, led to an exponential increase in investigations of these proteins and their corresponding metabolic pathways in Alzheimer disease (AD …
Identification of amyloid-beta and tau as the major protein components of senile plaques and neurofibrillary tangles, respectively, led to a …
Molecular pathogenesis of Alzheimer's disease: reductionist versus expansionist approaches.
Castellani RJ, Zhu X, Lee HG, Smith MA, Perry G. Castellani RJ, et al. Among authors: perry g, zhu x, lee hg, smith ma. Int J Mol Sci. 2009 Mar;10(3):1386-1406. doi: 10.3390/ijms10031386. Epub 2009 Mar 26. Int J Mol Sci. 2009. PMID: 19399255 Free PMC article. Review.
Alzheimer's disease (AD) is characterized clinically by dementia and pathologically by two hallmark lesions, senile plaques and neurofibrillary tangles. ...As such, AD pathology is therefore a host response rather than a manifestation of cytotox …
Alzheimer's disease (AD) is characterized clinically by dementia and pathologically by two hallmark lesions, senile plaques an …