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Results: 5

1.

Redox-dependent increases in glutathione reductase and exercise preconditioning: role of NADPH oxidase and mitochondria.

Frasier CR, Moukdar F, Patel HD, Sloan RC, Stewart LM, Alleman RJ, La Favor JD, Brown DA.

Cardiovasc Res. 2013 Apr 1;98(1):47-55. doi: 10.1093/cvr/cvt009. Epub 2013 Jan 22.

PMID:
23341578
[PubMed - indexed for MEDLINE]
Free Article
2.

Redox signaling (cross-talk) from and to mitochondria involves mitochondrial pores and reactive oxygen species.

Daiber A.

Biochim Biophys Acta. 2010 Jun-Jul;1797(6-7):897-906. doi: 10.1016/j.bbabio.2010.01.032. Epub 2010 Feb 1. Review.

PMID:
20122895
[PubMed - indexed for MEDLINE]
Free Article
3.

Oxidative stress and diabetic cardiovascular disorders: roles of mitochondria and NADPH oxidase.

Shen GX.

Can J Physiol Pharmacol. 2010 Mar;88(3):241-8. doi: 10.1139/Y10-018. Review.

PMID:
20393589
[PubMed - indexed for MEDLINE]
4.

Exercise preconditioning of the myocardium.

Kavazis AN.

Sports Med. 2009;39(11):923-35. doi: 10.2165/11317870-000000000-00000. Review.

PMID:
19827860
[PubMed - indexed for MEDLINE]
5.

[Glutathione system. II. Other enzymes, thiol-disulphide metabolism, inflammation and immunity, functions].

Kulinskiń≠ VI, Kolesnichenko LS.

Biomed Khim. 2009 Jul-Aug;55(4):365-79. Review. Russian.

PMID:
20000118
[PubMed - indexed for MEDLINE]

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