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Items: 6


Nitric oxide controls the immunopathology of tuberculosis by inhibiting NLRP3 inflammasome-dependent processing of IL-1β.

Mishra BB, Rathinam VA, Martens GW, Martinot AJ, Kornfeld H, Fitzgerald KA, Sassetti CM.

Nat Immunol. 2013 Jan;14(1):52-60. doi: 10.1038/ni.2474. Epub 2012 Nov 18.


Mycobacterium tuberculosis and the host cell inflammasome: a complex relationship.

Briken V, Ahlbrand SE, Shah S.

Front Cell Infect Microbiol. 2013 Oct 9;3:62. doi: 10.3389/fcimb.2013.00062. eCollection 2013. Review.


Mitochondria: Sovereign of inflammation?

Tschopp J.

Eur J Immunol. 2011 May;41(5):1196-202. doi: 10.1002/eji.201141436. Review.


The NLRP3 inflammasome in health and disease: the good, the bad and the ugly.

Menu P, Vince JE.

Clin Exp Immunol. 2011 Oct;166(1):1-15. doi: 10.1111/j.1365-2249.2011.04440.x. Epub 2011 Jul 15. Review.


Interleukin-1beta (IL-1beta) processing pathway.

Weber A, Wasiliew P, Kracht M.

Sci Signal. 2010 Jan 19;3(105):cm2. doi: 10.1126/scisignal.3105cm2. Review.


Biology of IL-27 and its role in the host immunity against Mycobacterium tuberculosis.

Abdalla AE, Li Q, Xie L, Xie J.

Int J Biol Sci. 2015 Jan 5;11(2):168-75. doi: 10.7150/ijbs.10464. eCollection 2015. Review.

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