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Results: 6

1.

CLMP is required for intestinal development, and loss-of-function mutations cause congenital short-bowel syndrome.

Van Der Werf CS, Wabbersen TD, Hsiao NH, Paredes J, Etchevers HC, Kroisel PM, Tibboel D, Babarit C, Schreiber RA, Hoffenberg EJ, Vekemans M, Zeder SL, Ceccherini I, Lyonnet S, Ribeiro AS, Seruca R, Te Meerman GJ, van Ijzendoorn SC, Shepherd IT, Verheij JB, Hofstra RM.

Gastroenterology. 2012 Mar;142(3):453-462.e3. doi: 10.1053/j.gastro.2011.11.038. Epub 2011 Dec 7.

PMID:
22155368
[PubMed - indexed for MEDLINE]
2.

Congenital short-bowel; a case study and review of the literature.

Schalamon J, Schober PH, Gallippi P, Matthyssens L, Höllwarth ME.

Eur J Pediatr Surg. 1999 Aug;9(4):248-50. Review.

PMID:
10532268
[PubMed - indexed for MEDLINE]
3.

The search for evolutionary developmental origins of aging in zebrafish: a novel intersection of developmental and senescence biology in the zebrafish model system.

Kishi S.

Birth Defects Res C Embryo Today. 2011 Sep;93(3):229-48. doi: 10.1002/bdrc.20217. Review.

PMID:
21932432
[PubMed - indexed for MEDLINE]
4.

Zebrafish as a model to study cardiac development and human cardiac disease.

Bakkers J.

Cardiovasc Res. 2011 Jul 15;91(2):279-88. doi: 10.1093/cvr/cvr098. Epub 2011 May 19. Review.

PMID:
21602174
[PubMed - indexed for MEDLINE]
Free PMC Article
5.

Zebrafish: a model for understanding diabetic complications.

Jörgens K, Hillebrands JL, Hammes HP, Kroll J.

Exp Clin Endocrinol Diabetes. 2012 Apr;120(4):186-7. doi: 10.1055/s-0032-1304565. Epub 2012 Mar 8. Review.

PMID:
22402952
[PubMed - indexed for MEDLINE]
6.

Stem cells as a potential future treatment of pediatric intestinal disorders.

Markel TA, Crisostomo PR, Lahm T, Novotny NM, Rescorla FJ, Tector J, Meldrum DR.

J Pediatr Surg. 2008 Nov;43(11):1953-63. doi: 10.1016/j.jpedsurg.2008.06.019. Review.

PMID:
18970924
[PubMed - indexed for MEDLINE]
Free PMC Article

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