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Items: 1 to 20 of 23

1.

T-cell receptor signaling enhances transcriptional elongation from latent HIV proviruses by activating P-TEFb through an ERK-dependent pathway.

Kim YK, Mbonye U, Hokello J, Karn J.

J Mol Biol. 2011 Jul 29;410(5):896-916. doi: 10.1016/j.jmb.2011.03.054.

2.

New insights into the control of HIV-1 transcription: when Tat meets the 7SK snRNP and super elongation complex (SEC).

He N, Zhou Q.

J Neuroimmune Pharmacol. 2011 Jun;6(2):260-8. doi: 10.1007/s11481-011-9267-6. Epub 2011 Mar 1. Review.

3.

RNA-driven cyclin-dependent kinase regulation: when CDK9/cyclin T subunits of P-TEFb meet their ribonucleoprotein partners.

Michels AA, Bensaude O.

Biotechnol J. 2008 Aug;3(8):1022-32. doi: 10.1002/biot.200800104. Review.

PMID:
18655042
4.

Molecular mechanisms of HIV latency.

Cary DC, Fujinaga K, Peterlin BM.

J Clin Invest. 2016 Feb;126(2):448-54. doi: 10.1172/JCI80565. Epub 2016 Jan 5. Review.

PMID:
26731470
5.

The molecular biology of HIV latency: breaking and restoring the Tat-dependent transcriptional circuit.

Karn J.

Curr Opin HIV AIDS. 2011 Jan;6(1):4-11. doi: 10.1097/COH.0b013e328340ffbb. Review.

6.

Regulation of TAK/P-TEFb in CD4+ T lymphocytes and macrophages.

Rice AP, Herrmann CH.

Curr HIV Res. 2003 Oct;1(4):395-404. Review. Erratum in: Curr HIV Res. 2004 Apr;2(2):205.

PMID:
15049426
7.

Transcriptional control of HIV latency: cellular signaling pathways, epigenetics, happenstance and the hope for a cure.

Mbonye U, Karn J.

Virology. 2014 Apr;454-455:328-39. doi: 10.1016/j.virol.2014.02.008. Epub 2014 Feb 22. Review.

8.

CYCLINg through transcription: posttranslational modifications of P-TEFb regulate transcription elongation.

Cho S, Schroeder S, Ott M.

Cell Cycle. 2010 May;9(9):1697-705. Epub 2010 May 29. Review.

9.

Targeting HIV transcription: the quest for a functional cure.

Mousseau G, Mediouni S, Valente ST.

Curr Top Microbiol Immunol. 2015;389:121-45. doi: 10.1007/82_2015_435. Review.

10.

Mechanism and factors that control HIV-1 transcription and latency activation.

Liu RD, Wu J, Shao R, Xue YH.

J Zhejiang Univ Sci B. 2014 May;15(5):455-65. doi: 10.1631/jzus.B1400059. Review.

11.

Brd4 and HEXIM1: multiple roles in P-TEFb regulation and cancer.

Chen R, Yik JH, Lew QJ, Chao SH.

Biomed Res Int. 2014;2014:232870. doi: 10.1155/2014/232870. Epub 2014 Jan 29. Review.

12.

Expanding role of cyclin dependent kinases in cytokine inducible gene expression.

Brasier AR.

Cell Cycle. 2008 Sep 1;7(17):2661-6. Epub 2008 Sep 12. Review.

13.

Anti-viral opportunities during transcriptional activation of latent HIV in the host chromatin.

Mujtaba S, Zhou MM.

Methods. 2011 Jan;53(1):97-101. doi: 10.1016/j.ymeth.2010.09.001. Epub 2010 Sep 7. Review.

14.

Lost in transcription: molecular mechanisms that control HIV latency.

Taube R, Peterlin M.

Viruses. 2013 Mar 21;5(3):902-27. doi: 10.3390/v5030902. Review.

15.

HEXIM1 and the control of transcription elongation: from cancer and inflammation to AIDS and cardiac hypertrophy.

Dey A, Chao SH, Lane DP.

Cell Cycle. 2007 Aug 1;6(15):1856-63. Epub 2007 Jun 6. Review.

PMID:
17671421
16.

The multi-tasking P-TEFb complex.

Brès V, Yoh SM, Jones KA.

Curr Opin Cell Biol. 2008 Jun;20(3):334-40. doi: 10.1016/j.ceb.2008.04.008. Epub 2008 May 29. Review.

17.

Role of noncoding RNAs in the regulation of P-TEFb availability and enzymatic activity.

Napolitano G, Lania L, Majello B.

Biomed Res Int. 2014;2014:643805. doi: 10.1155/2014/643805. Epub 2014 Feb 19. Review.

18.

The multifactorial nature of HIV-1 latency.

Lassen K, Han Y, Zhou Y, Siliciano J, Siliciano RF.

Trends Mol Med. 2004 Nov;10(11):525-31. Review.

PMID:
15519278
19.

Megakaryocytic programming by a transcriptional regulatory loop: A circle connecting RUNX1, GATA-1, and P-TEFb.

Goldfarb AN.

J Cell Biochem. 2009 Jun 1;107(3):377-82. doi: 10.1002/jcb.22142. Review.

20.

Viral-host interactions that control HIV-1 transcriptional elongation.

Lu H, Li Z, Xue Y, Zhou Q.

Chem Rev. 2013 Nov 13;113(11):8567-82. doi: 10.1021/cr400120z. Epub 2013 Jun 24. Review. No abstract available.

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