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Items: 1 to 20 of 37

1.

Inhibition of MALT1 protease activity is selectively toxic for activated B cell-like diffuse large B cell lymphoma cells.

Ferch U, Kloo B, Gewies A, Pfänder V, Düwel M, Peschel C, Krappmann D, Ruland J.

J Exp Med. 2009 Oct 26;206(11):2313-20. doi: 10.1084/jem.20091167. Epub 2009 Oct 19. Erratum in: J Exp Med. 2009 Nov 23;206(12):2851.

2.

Molecular pathways: targeting MALT1 paracaspase activity in lymphoma.

Fontán L, Melnick A.

Clin Cancer Res. 2013 Dec 15;19(24):6662-8. doi: 10.1158/1078-0432.CCR-12-3869. Epub 2013 Sep 4. Review.

3.

The roles of CARMA1, Bcl10, and MALT1 in antigen receptor signaling.

Lin X, Wang D.

Semin Immunol. 2004 Dec;16(6):429-35. Review.

PMID:
15541657
4.

NF-kappaB signaling in lymphocytes: a new cast of characters.

Lucas PC, McAllister-Lucas LM, Nunez G.

J Cell Sci. 2004 Jan 1;117(Pt 1):31-9. Review.

5.

TCR-induced NF-kappaB activation: a crucial role for Carma1, Bcl10 and MALT1.

Thome M, Tschopp J.

Trends Immunol. 2003 Aug;24(8):419-24. Review. No abstract available.

PMID:
12909454
6.

Recent prognostic factors in diffuse large B-cell lymphoma indicate NF-kappaB pathway as a target for new therapeutic strategies.

Pavan A, Spina M, Canzonieri V, Sansonno S, Toffoli G, De Re V.

Leuk Lymphoma. 2008 Nov;49(11):2048-58. doi: 10.1080/10428190802444176. Review.

PMID:
19021048
7.
8.

The Bcl10/Malt1 signaling pathway as a drug target in lymphoma.

Jost P, Peschel C, Ruland J.

Curr Drug Targets. 2006 Oct;7(10):1335-40. Review.

PMID:
17073595
9.

Determining the destiny of NF-kappa B after TCR ligation: it's CARMA1.

Bunnell SC.

Mol Interv. 2002 Oct;2(6):356-60, 338. Review.

PMID:
14993411
10.

Protease activity of MALT1: a mystery unravelled.

Kirchhofer D, Vucic D.

Biochem J. 2012 Jun 1;444(2):e3-5. doi: 10.1042/BJ20120414. Review.

PMID:
22574776
11.

The CARD11-BCL10-MALT1 (CBM) signalosome complex: Stepping into the limelight of human primary immunodeficiency.

Turvey SE, Durandy A, Fischer A, Fung SY, Geha RS, Gewies A, Giese T, Greil J, Keller B, McKinnon ML, Neven B, Rozmus J, Ruland J, Snow AL, Stepensky P, Warnatz K.

J Allergy Clin Immunol. 2014 Aug;134(2):276-84. doi: 10.1016/j.jaci.2014.06.015. Review.

12.

Malt1 and cIAP2-Malt1 as effectors of NF-kappaB activation: kissing cousins or distant relatives?

Kingeter LM, Schaefer BC.

Cell Signal. 2010 Jan;22(1):9-22. doi: 10.1016/j.cellsig.2009.09.033. Epub 2009 Sep 19. Review.

13.

Antigen receptor signaling to NF-kappaB via CARMA1, BCL10, and MALT1.

Thome M, Charton JE, Pelzer C, Hailfinger S.

Cold Spring Harb Perspect Biol. 2010 Sep;2(9):a003004. doi: 10.1101/cshperspect.a003004. Epub 2010 Aug 4. Review.

14.

Gene expression profiling of diffuse large B-cell lymphoma.

Rosenwald A, Staudt LM.

Leuk Lymphoma. 2003;44 Suppl 3:S41-7. Review.

PMID:
15202524
15.

B-cell receptor signaling in diffuse large B-cell lymphoma.

Young RM, Shaffer AL 3rd, Phelan JD, Staudt LM.

Semin Hematol. 2015 Apr;52(2):77-85. doi: 10.1053/j.seminhematol.2015.01.008. Epub 2015 Jan 15. Review.

PMID:
25805587
16.

MALT1 protease: a new therapeutic target in B lymphoma and beyond?

McAllister-Lucas LM, Baens M, Lucas PC.

Clin Cancer Res. 2011 Nov 1;17(21):6623-31. doi: 10.1158/1078-0432.CCR-11-0467. Epub 2011 Aug 25. Review.

17.

The CBM signalosome: potential therapeutic target for aggressive lymphoma?

Yang C, David L, Qiao Q, Damko E, Wu H.

Cytokine Growth Factor Rev. 2014 Apr;25(2):175-83. doi: 10.1016/j.cytogfr.2013.12.008. Epub 2013 Dec 24. Review.

18.

Regulation of NF-κB signaling by caspases and MALT1 paracaspase.

Staal J, Bekaert T, Beyaert R.

Cell Res. 2011 Jan;21(1):40-54. doi: 10.1038/cr.2010.168. Epub 2010 Nov 30. Review.

19.

NF-κB signaling pathways regulated by CARMA family of scaffold proteins.

Blonska M, Lin X.

Cell Res. 2011 Jan;21(1):55-70. doi: 10.1038/cr.2010.182. Epub 2010 Dec 28. Review.

20.

From MALT lymphoma to the CBM signalosome: three decades of discovery.

Rosebeck S, Rehman AO, Lucas PC, McAllister-Lucas LM.

Cell Cycle. 2011 Aug 1;10(15):2485-96. Epub 2011 Aug 1. Review.

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