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Results: 13

Cited In for PubMed (Select 9135004)


Basic transcription factor 3 is involved in gastric cancer development and progression.

Liu Q, Zhou JP, Li B, Huang ZC, Dong HY, Li GY, Zhou K, Nie SL.

World J Gastroenterol. 2013 Jul 28;19(28):4495-503. doi: 10.3748/wjg.v19.i28.4495.


The role of the DNA damage response kinase ataxia telangiectasia mutated in neuroprotection.

Marinoglou K.

Yale J Biol Med. 2012 Dec;85(4):469-80. Epub 2012 Dec 13. Review.


Redundant and nonredundant functions of ATM and H2AX in αβ T-lineage lymphocytes.

Yin B, Lee BS, Yang-Iott KS, Sleckman BP, Bassing CH.

J Immunol. 2012 Aug 1;189(3):1372-9. doi: 10.4049/jimmunol.1200829. Epub 2012 Jun 22.


Induction and utilization of an ATM signaling pathway by polyomavirus.

Dahl J, You J, Benjamin TL.

J Virol. 2005 Oct;79(20):13007-17.


Phosphorylation of serine 18 regulates distinct p53 functions in mice.

Sluss HK, Armata H, Gallant J, Jones SN.

Mol Cell Biol. 2004 Feb;24(3):976-84.


Brostallicin (PNU-166196)--a new DNA minor groove binder that retains sensitivity in DNA mismatch repair-deficient tumour cells.

Fedier A, Fowst C, Tursi J, Geroni C, Haller U, Marchini S, Fink D.

Br J Cancer. 2003 Oct 20;89(8):1559-65.


ATM-dependent expression of the insulin-like growth factor-I receptor in a pathway regulating radiation response.

Peretz S, Jensen R, Baserga R, Glazer PM.

Proc Natl Acad Sci U S A. 2001 Feb 13;98(4):1676-81. Epub 2001 Feb 6.


ATM protein and p53-serine 15 phosphorylation in ataxia-telangiectasia (AT) patients and at heterozygotes.

Delia D, Mizutani S, Panigone S, Tagliabue E, Fontanella E, Asada M, Yamada T, Taya Y, Prudente S, Saviozzi S, Frati L, Pierotti MA, Chessa L.

Br J Cancer. 2000 Jun;82(12):1938-45.


Rapid ATM-dependent phosphorylation of MDM2 precedes p53 accumulation in response to DNA damage.

Khosravi R, Maya R, Gottlieb T, Oren M, Shiloh Y, Shkedy D.

Proc Natl Acad Sci U S A. 1999 Dec 21;96(26):14973-7.


Radiation-induced G1 arrest is not defective in fibroblasts from Li-Fraumeni families without TP53 mutations.

Boyle JM, Greaves MJ, Camplejohn RS, Birch JM, Roberts SA, Varley JM.

Br J Cancer. 1999 Apr;79(11-12):1657-64.


Atm is dispensable for p53 apoptosis and tumor suppression triggered by cell cycle dysfunction.

Liao MJ, Yin C, Barlow C, Wynshaw-Boris A, van Dyke T.

Mol Cell Biol. 1999 Apr;19(4):3095-102.

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