Sort by

Send to

Choose Destination

Links from PubMed

Items: 11


Targeted Knockdown of the Kinetochore Protein D40/Knl-1 Inhibits Human Cancer in a p53 Status-Independent Manner.

Urata YN, Takeshita F, Tanaka H, Ochiya T, Takimoto M.

Sci Rep. 2015 Sep 8;5:13676. doi: 10.1038/srep13676.


Effects and interactions of MiR-577 and TSGA10 in regulating esophageal squamous cell carcinoma.

Yuan X, He J, Sun F, Gu J.

Int J Clin Exp Pathol. 2013 Nov 15;6(12):2651-67. eCollection 2013.


Paradoxical suppression of cellular senescence by p53.

Demidenko ZN, Korotchkina LG, Gudkov AV, Blagosklonny MV.

Proc Natl Acad Sci U S A. 2010 May 25;107(21):9660-4. doi: 10.1073/pnas.1002298107. Epub 2010 May 10.


Antisense MDM2 enhances E2F1-induced apoptosis and the combination sensitizes androgen-sensitive [corrected] and androgen-insensitive [corrected] prostate cancer cells to radiation.

Udayakumar TS, Hachem P, Ahmed MM, Agrawal S, Pollack A.

Mol Cancer Res. 2008 Nov;6(11):1742-54. doi: 10.1158/1541-7786.MCR-08-0102. Erratum in: Mol Cancer Res. 2008 Dec;6(12):1957.


Cell cycle control as a basis for cancer chemoprevention through dietary agents.

Meeran SM, Katiyar SK.

Front Biosci. 2008 Jan 1;13:2191-202. Review.


Roles of the Raf/MEK/ERK pathway in cell growth, malignant transformation and drug resistance.

McCubrey JA, Steelman LS, Chappell WH, Abrams SL, Wong EW, Chang F, Lehmann B, Terrian DM, Milella M, Tafuri A, Stivala F, Libra M, Basecke J, Evangelisti C, Martelli AM, Franklin RA.

Biochim Biophys Acta. 2007 Aug;1773(8):1263-84. Epub 2006 Oct 7. Review.


Direct repression of FLIP expression by c-myc is a major determinant of TRAIL sensitivity.

Ricci MS, Jin Z, Dews M, Yu D, Thomas-Tikhonenko A, Dicker DT, El-Deiry WS.

Mol Cell Biol. 2004 Oct;24(19):8541-55.


Sodium butyrate and tributyrin induce in vivo growth inhibition and apoptosis in human prostate cancer.

Kuefer R, Hofer MD, Altug V, Zorn C, Genze F, Kunzi-Rapp K, Hautmann RE, Gschwend JE.

Br J Cancer. 2004 Jan 26;90(2):535-41.

Items per page

Supplemental Content

Write to the Help Desk